Titlebook: Interactive Phenomena in the Cardiac System; Samuel Sideman,Rafael Beyar Book 1993 The Editor(s) (if applicable) and The Author(s), under

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Neuromodulation of Calcium Current by Extracellular ATP in Isolated Ventricular Myocytesally isolated myocytes from the right ventricles of ferrets. Extracellular ATP decreased the peak amplitude of I. in a time- and concentration-dependent manner. The concentration-response relationship for ATP inhibition of I. was well described by a conventional Michaelis-Menten relationship with a

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Intracellular Calcium and Myocardial Function During Ischemia mechanical performance could, in principle, be caused either by reduced intracellular calcium release or by reduced responsiveness of the myofibrillar proteins to calcium. It is now known that intracellular calcium rises during ischemia and that the early decline in mechanical performance is caused

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Intracellular Signaling in Vascular Smooth Musclewed. Recent studies show that although changes in cytoplasmic Ca. play the major role in regulating smooth muscle contraction, agonists can also significantly affect the contractile state by modifying Ca.-sensitivity. Inhibition of myosin light chain kinase or myosin light chain phosphatase leads to

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Intramyocardial Mechanical States: Vessel-Interstitium-Muscle InterfaceCoronary circulation was found to be strongly influenced by passive and active properties of the muscle surrounding the coronary microvasculature. Selective remodeling of the extracellular collagen-matrix and partial uncoupling of the intramyocardial structure(s) cause profound functional alteration

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