Titlebook: Inflammatory Dermatopathology; A Pathologist‘s Surv Steven D. Billings,Jenny Cotton Book 2016Latest edition Springer International Publishi

导师

Introduction,y can border on the impenetrable. So often a specific diagnosis is elusive. As a result we often rely on diagnoses such as non-specific chronic dermatitis. Therein lies the problem. There is nothing a dermatologist or other clinician hates more than the diagnosis of “nonspecific chronic dermatitis.”

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lipids

Perivascular Dermatitis,ely restricted to the superficial, or superficial and deep dermis around blood vessels. Not infrequently, there may be some overlap with the superficial, and superficial and deep perivascular patterns. To the consternation of many pathologists, initial perusal of a skin biopsy demonstrating a superf

ostracize

等级的上升

FEMUR

Palisading Granulomatous Dermatitis,f altered collagen. Classically, the inflammatory infiltrate surrounds the zones of altered collagen in a wall-like or fence-like fashion, hence the term “palisading.” The classic entities in this differential diagnosis include granuloma annulare, necrobiosis lipoidica, rheumatoid nodule, and palisa

archetype

Bullous Dermatitis,ich blister formation is the primary manifestation rather than blisters as a secondary phenomenon (i.e., blisters secondary to contact dermatitis, as discussed in Chap. .). An understanding of the concept of acantholysis is paramount to any discussion of the intraepidermal vesicular disorders. Acant

莎草

Panniculitis,nicians and pathologists alike for several reasons. First, clinical overlap is common amongst the diseases. Second, there are often sampling issues, including inadequate, superficial biopsies, which preclude optimal evaluation of the fat. Finally, as in all cutaneous inflammatory disease, the pannic

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