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Titlebook: Inflammation, Infection, and Microbiome in Cancers; Evidence, Mechanisms Jun Sun Book 2021 The American Physiological Society 2021 Cancer p

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发表于 2025-3-21 17:36:46 | 显示全部楼层 |阅读模式
书目名称Inflammation, Infection, and Microbiome in Cancers
副标题Evidence, Mechanisms
编辑Jun Sun
视频video
概述Highlights connections between infection, inflammation and various cancers.Offers insights into the progress of immune therapy and stem cell research in cancer.Discusses emerging roles of the microbio
丛书名称Physiology in Health and Disease
图书封面Titlebook: Inflammation, Infection, and Microbiome in Cancers; Evidence, Mechanisms Jun Sun Book 2021 The American Physiological Society 2021 Cancer p
描述.This book offers a summary and discussion of the advances of inflammation and infection in various cancers. The authors cover the classically known virus infections in cancer, novel roles of other pathogens (e.g. bacteria and fungi), as well as biomarkers for diagnosis and therapy. Further, the chapters highlight the progress of immune therapy, stem cells and the role of the microbiome in the pathophysiology of cancers..Readers will gain insights into complex microbial communities, that inhabit most external human surfaces and play a key role in health and disease. Perturbations of host-microbe interactions often lead to altered host responses that can promote cancer development. Thus, this book highlights emerging roles of the microbiome in pathogenesis of cancers and outcome of therapy. The focus is on mechanistic concepts that underlie the complex relationships between host and microbes. Approaches that can inhibit infection, suppress chronic inflammation and reverse the dysbiosis are discussed, as a means for restoring the balance between host and microbes..This comprehensive work will be beneficial to researchers and students interested in infectious diseases, microbiome, and
出版日期Book 2021
关键词Cancer pathogens; Microbiome; microbe Infection; Inflammation; cancer biomarkers
版次1
doihttps://doi.org/10.1007/978-3-030-67951-4
isbn_softcover978-3-030-67953-8
isbn_ebook978-3-030-67951-4Series ISSN 2625-252X Series E-ISSN 2625-2538
issn_series 2625-252X
copyrightThe American Physiological Society 2021
The information of publication is updating

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发表于 2025-3-21 22:05:27 | 显示全部楼层
Commensal Microbes and Their Metabolites: Influence on Host Pathways in Health and Cancer,tabolic process. In this chapter, we have discussed the major microbial metabolites that dominate the metabolic landscape of gut microbiota (the cancer microbiome) and influence cancer pathogenesis and chemotherapeutic efficacy.
发表于 2025-3-22 02:17:36 | 显示全部楼层
Mitochondrial Regulation of Inflammation in Cancer,ival of tumor cells. Current pharmacological agents seek to exploit this process by targeting either the mitochondria or downstream targets of the mitochondria which promote inflammation. This chapter delves into the origins of mitochondrial dysfunction and the corresponding signaling pathways that regulate inflammation in cancer.
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Biomarkers of Esophageal Cancers and Precancerous Lesions,ncer and precancerous lesions that have already been in clinical application as well as those that are in different stages of discovery and validation. In addition, we briefly introduce the microbiome and other less conventional biomarkers of esophageal cancers.
发表于 2025-3-22 17:44:55 | 显示全部楼层
Epithelial and Immune Cell Responses to , That Shape the Gastric Tumor Microenvironment,y response to . infection and immune cell-epithelial crosstalk. The review focuses on reporting the mechanisms by which the bacterium modulates the host’s innate and adaptive immune response and the gastric epithelium as part of a strategy to create an immunosuppressive microenvironment that ultimately leads to gastric cancer.
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Autophagy and Cancer: Current Biology and Drug Development,lective autophagy of mitochondria, called mitophagy, in tumorigenesis. Understanding the roles of autophagy and mitophagy in cancer stem cells, cancer immunosurveillance, cancer metabolism, and iron homeostasis will influence development of emerging anti-cancer therapeutics targeting autophagy.
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