找回密码
 To register

QQ登录

只需一步,快速开始

扫一扫,访问微社区

Titlebook: Inflammation Protocols; Paul G. Winyard,Derek A. Willoughby Book 2003 Humana Press 2003

[复制链接]
查看: 25508|回复: 52
发表于 2025-3-21 19:47:09 | 显示全部楼层 |阅读模式
书目名称Inflammation Protocols
编辑Paul G. Winyard,Derek A. Willoughby
视频videohttp://file.papertrans.cn/465/464669/464669.mp4
概述Includes supplementary material:
丛书名称Methods in Molecular Biology
图书封面Titlebook: Inflammation Protocols;  Paul G. Winyard,Derek A. Willoughby Book 2003 Humana Press 2003
描述Inflammation has been described as the basis of many pathologies of human disease. When one considers the updated signs of inflammation, they would be vasodilation, cell migration, and, in the case of chronic inflam- tion, cell proliferation, often with an underlying autoimmune basis. Gen- ally, inflammation may be divided into acute, chronic, and autoimmune, - though the editors believe that most, if not all, chronic states are often the result of an autoimmune response to an endogenous antigen. Thus, a proper understanding of the inflammatory basis may provide clues to new therap- tic targets not only in classical inflammatory diseases, but atherosclerosis, cancer, and ischemic heart disease as well. The lack of advances in classical inflammatory diseases, such as rh- matoid arthritis, may in part arise from a failure to classify the disease into different forms. That different forms exist is exemplified in patients with d- fering responses to existing antiinflammatory drugs, ranging from nonresponders to very positive responders for a particular nonsteroidal an- inflammatory drug (NSAID). Though researchers have progressively unr- eled the mechanisms, the story is far from compl
出版日期Book 2003
版次1
doihttps://doi.org/10.1385/1592593747
isbn_softcover978-1-61737-282-7
isbn_ebook978-1-59259-374-3Series ISSN 1064-3745 Series E-ISSN 1940-6029
issn_series 1064-3745
copyrightHumana Press 2003
The information of publication is updating

书目名称Inflammation Protocols影响因子(影响力)




书目名称Inflammation Protocols影响因子(影响力)学科排名




书目名称Inflammation Protocols网络公开度




书目名称Inflammation Protocols网络公开度学科排名




书目名称Inflammation Protocols被引频次




书目名称Inflammation Protocols被引频次学科排名




书目名称Inflammation Protocols年度引用




书目名称Inflammation Protocols年度引用学科排名




书目名称Inflammation Protocols读者反馈




书目名称Inflammation Protocols读者反馈学科排名




单选投票, 共有 1 人参与投票
 

0票 0.00%

Perfect with Aesthetics

 

0票 0.00%

Better Implies Difficulty

 

1票 100.00%

Good and Satisfactory

 

0票 0.00%

Adverse Performance

 

0票 0.00%

Disdainful Garbage

您所在的用户组没有投票权限
发表于 2025-3-21 23:03:42 | 显示全部楼层
发表于 2025-3-22 02:25:50 | 显示全部楼层
发表于 2025-3-22 04:55:24 | 显示全部楼层
1064-3745 pdated signs of inflammation, they would be vasodilation, cell migration, and, in the case of chronic inflam- tion, cell proliferation, often with an underlying autoimmune basis. Gen- ally, inflammation may be divided into acute, chronic, and autoimmune, - though the editors believe that most, if no
发表于 2025-3-22 12:26:11 | 显示全部楼层
发表于 2025-3-22 15:45:39 | 显示全部楼层
Inflammatory Joint Diseaseand hormonal influences (.–.). Moreover, the mechanisms responsible for the development of SCW-arthritis are susceptible to regulation by pharmacologic agents and biological response modifiers and as such, this model is considered a valid preclinical model of RA.
发表于 2025-3-22 17:46:06 | 显示全部楼层
Key Stages in the Acute Inflammatory Response and Their Relevance as Therapeutic Targetsy response. For the reader who is unfamiliar with the field of inflammation, it is perhaps helpful to summarize and contextualize some of the key events of the inflammatory response, as it is these that may be reproduced in the form of in vitro model systems by using the protocols that follow.
发表于 2025-3-22 21:44:20 | 显示全部楼层
Screening for Inhibitors of Transcription Factors Using Luciferase Reporter Gene Expression in Trans of TF function offers an innovative approach to disease therapies. TF modulation holds particular promise in diseases where several proteins contribute simultaneously to the pathology, such as in autoimmune disease, where a plethora of cytokines, destructive enzymes, and cell-adhesion molecules combine to enhance disease progression.
发表于 2025-3-23 02:00:52 | 显示全部楼层
发表于 2025-3-23 07:30:05 | 显示全部楼层
Book 2003 vasodilation, cell migration, and, in the case of chronic inflam- tion, cell proliferation, often with an underlying autoimmune basis. Gen- ally, inflammation may be divided into acute, chronic, and autoimmune, - though the editors believe that most, if not all, chronic states are often the result
 关于派博传思  派博传思旗下网站  友情链接
派博传思介绍 公司地理位置 论文服务流程 影响因子官网 吾爱论文网 大讲堂 北京大学 Oxford Uni. Harvard Uni.
发展历史沿革 期刊点评 投稿经验总结 SCIENCEGARD IMPACTFACTOR 派博系数 清华大学 Yale Uni. Stanford Uni.
QQ|Archiver|手机版|小黑屋| 派博传思国际 ( 京公网安备110108008328) GMT+8, 2025-8-15 09:22
Copyright © 2001-2015 派博传思   京公网安备110108008328 版权所有 All rights reserved
快速回复 返回顶部 返回列表