Titlebook: Industrial and Environmental Xenobiotics; Metabolism and Pharm Ivan Gut,Miroslav Cikrt,Gabriel L. Plaa Conference proceedings 1981 Springer

Anthology

Rat Liver and Kidney Gluconeogenesis After Acute Intoxication with Carbon Tetrachloridefructose bisphosphatase, phosphoenolpyruvate carboxykinase and lactate dehydrogenase have been described among the affected enzymes (Serban and Serban 1973; Taketa et al. 1976; Kamp and Hornbrook 1977; Faus et al. 1978), hepatic gluconeogenesis being impaired under these circumstances (Faus et al. 1

delusion

Evaluation of Hepatic Mixed Function Oxidase (MFO) Systems After Administration of Allylisopropylacestered to animals, most of these chemicals either induce or have little or no effect on MFO systems, but a few produce marked losses. Allylisopropylacetamide (AIA), CCl., CS. and polyriboinosinic acid·polyribocytidylic acid (poly rI·rC) are prototypes of xenobiotics which destroy MFO systems by diff

exceptional

行为

commonsense

间谍活动

Inorganic Cations Toxicity-Application of QSAR Analysisic or organic ligands. “Properties” of “the cation” will be accordingly different depending on properties of the complex as a whole and the difference might in some cases be very large. This contribution contains our results concerning both aspects.

Fester

破裂

0172-6625 ­ vironmental Xenobiotics, held in Prague, 1980, and some contributions by those who could not come. The first aim of the meeting was to fol­ low the tradition set up by the first conference in 1977. Again, we in­ vited biochemists, pharmacologists, and toxicologists from both East and West, who wer

CHAFE

Metabolic Factors in the Distribution and Half Time of Mercury After Exposure to Different Mercurialof mercury are rapidly converted to sulphydryl-bound mercuric mercury. Even short chain alkylmercurials — though at a very much slower rate — are decomposed to mercuric mercury and before decomposition the intact methylmercury — like inorganic mercury — is mostly bound to sulphydryl binding sites.

gerontocracy

Pulmonary Toxicity of Carbon Tetrachlorideecknagel and Glende 1973) or by binding covalently to essential cellular macromolecules (e.g., see Reynolds 1967; Rao and Recknagel 1969; Castro and Gomez 1972; Recknagel and Glende 1973; Villarruel et al. 1977) or possibly by both mechanisms.