Titlebook: Immunopharmacology and Inflammation; Carlo Riccardi,Francesca Levi-Schaffer,Ekaterini T Book 2018 Springer International Publishing AG, pa

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Adaptive Immunity of Airway Inflammation in Asthmaropriate activation of these immune systems in the respiratory mucosa results in chronic inflammatory airways disease such as asthma. Adaptive immunity is stimulated for example by allergen exposure that activates T and B lymphocytes leading to IgE production and influx of eosinophilic granulocytes

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Neural Regulation of Inflammation: Pharmacological Mechanisms and Therapeutic Perspectiveshugely increased, and the relevance of the neuroimmune network in health and disease is now established, providing novel and unanticipated opportunities for the modulation of the immune response by means of conventional neural targets. Primary and secondary lymphoyd organs are extensively innervated

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Pro-resolving Mediatorsf inflammation. It has been argued that uncontrolled inflammation may be the basis of a variety of chronic inflammatory and autoimmune diseases. The resolution of inflammation is an active process coordinated by the production of proresolving mediators. The release of proresolving mediators prevents

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Innate Immunity in Inflammationes the complexity of the immune system, at the same time pointing out the challenge for effective and safe immunopharmacological intervention. A wide variety of clinically relevant drugs are currently used in the treatment of human inflammatory and immune-system associated disorders. Classical thera

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Immune Mechanisms in Atherosclerosis and Potential for Immunomodulatory Therapiestion and development. Recent clinical trial results confirm the feasibility of targeting immune pathways for the therapeutic control of the pathology. In this chapter, we discuss the key immune-inflammatory mechanisms involved in atherosclerosis development and progression and plaque destabilization

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Regulation of Immunity and Disease by the IL-1 Receptor Family Members IL-1R2 and IL-1R8ugh ligands with antagonistic or anti-inflammatory activity, or decoy and negative regulatory receptors. IL-1R2 and IL-1R8 (also known as SIGIRR) are members of the ILR family acting as negative regulators of the IL-1 system. IL-1R2 binds IL-1 and the accessory protein IL-1RAcP without activating si