Titlebook: Immunology of Fungal Infections; Gordon D. Brown,Mihai G. Netea Book 2007 Springer Science+Business Media B.V. 2007 AIDS.Fungal Infections

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Complement in fungal infections and complement evasion strategies, enables a longer survival of organ transplantation recipients, or due to better treatment options for chronic infections or neoplastic diseases. These improved therapeutic approaches will keep more patients for longer periods in immunocompromised states, where they are in particular susceptible to

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Recognition of fungal pathogens by Toll-like receptorsns (PAMPs) by TLRs, either alone or in heterodimerization with other TLR or non-TLR receptors, induces signals responsible for the activation of innate immune response. Recent studies have demonstrated a crucial involvement of TLRs in the recognition of fungal pathogens such as . and .. By studying

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Fungal ß-Glucans and their Receptorsrates is thought to occur via multiple receptors and at least four different receptors have been identified, including Dectin-1, complement receptor 3, lactosylceramide, and scavenger receptors. There is growing evidence that ß-glucan recognition is an important component of anti-fungal immunity, bu

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Detection of Fungi by Mannose-based Recognition Receptorsal viability. They constitute Pathogen-Associated-Molecular-Patterns (PAMPs) that can be detected by specific recognition receptors expressed by mammalian antigen-presenting cells. Fungal mannosylation, the mammalian membrane receptors that have evolved to detect fungal mannose groups and the immune

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Pneumocystise advent of highly active anti-retroviral therapy (HAART), . remains the number one AIDS-defining illness. The host response to . involves a fine balance between cellular activation leading to organism clearance and lung injury associated with the immune response. This review will focus on the innat

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