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Titlebook: Immunodermatology; Irma N. Gigli,Peter A. Miescher,Hans J. Müller-Ebe Book 1983 Springer-Verlag Berlin Heidelberg 1983 Dermatitis.Hautkran

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Elaine L. Alexander,Thomas T. Provostependence of all these quantities on the frame of reference. In many cases, when such an accurate description of motion is too complex, it is convenient to substitute the real body with an ideal body for which the analysis of the preceding characteristics is simpler, provided that the kinematic desc
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s who are interested in the applications of statistical methods to plasma physics. Thus it provides also concise information on experimental aspects of fusion-oriented plasma physics. In view of the first three basic chapters it can be fruitfully used by students majoring in probability theory and s
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Introduction, such as contact hypersensitivity and urticeria. Gradually, the spectrum of immunological conditions involving the skin increased, encompassing diseases such as Dermatitis Herpetiformis, Erythema Multiforme, Cutaneous Bullous Disorders and Phototoxic Reactions.
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Immunologically Mediated Epidermal Cell Injury,es that make up the epidermis (keratinocytes, melanocytes, and Langerhans cells) serve different protective functions. The vast majority of epidermal cells are keratinocytes (Fig. 1). This ectodermally derived cell type undergoes a process of terminal differentiation in which cells that are produced
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Dermatitis Herpetiformis,edge about DH has been attained over the past 15 years. Stimulated by the discoveries of Cormane [3] and Van der Meer [45] that IgA is deposited in the skin of these patients, and the discovery of Marks et al. [25] that there is an associated gastrointestinal abnormality, intense investigation has b
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Erythema Multiforme: Clinical Spectrum and Immunopathogenesis,for recurrence, and sometimes distinctive clinical appearance. Although initially differentiated from other cutaneous erythemas by Hebra in 1866 [6], the full characterization of erythema multiforme has been slow, due to the tendency to include many poorly characterized toxic erythematous eruptions
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Biology of the Mast Cell and Its Role in Cutaneous Inflammation, mucosal and cutaneous surfaces and about venules places the mast cell in an ideal location for function in inflammation and host defense.This review will consider the biology of the tissue mast cell so that its function may be discussed in the chapters on urticaria and bullous diseases.
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Mast Cell-Dependent Effector Pathways and Cutaneous Bullous Disorders,ervations led to the hypothesis that these cutaneous blistering disorders may be autoimmune diseases in which antibody and complement participate to mediate the development of clinical lesions. Recent observations suggest that mast cell-initiated effector pathways may at least play a role in the pat
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Physical Urticaria/Angioedema as an Experimental Model of Acute and Chronic Inflammation in Human S. The eliciting stimuli include trauma, pressure, vibration, light, cold, heat, and water. The IgE-dependent nature of certain forms of physical urticaria/angioedema has been demonstrated by passive transfer of the reaction with serum to normal individuals (Prausnitz-Küstner reaction). Urticaria app
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