Titlebook: Hypoxia; Into the Next Millen Robert C. Roach,Peter D. Wagner,Peter H. Hackett Book 1999 The Editor(s) (if applicable) and The Author(s), u

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angiography

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https://doi.org/10.1007/978-3-658-24114-8, this is still unproven. Symptoms of HACE are related to increased intracranial pressure (ICP), and death is from brain herniation. Treatment is directed both to lowering ICP by reducing the volume of intracranial contents, and to stopping the vasogenic leak..Evidence is accumulating that establish

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https://doi.org/10.1007/978-3-531-91150-2oted to the mechanism for the increase in arterial oxygen saturation on the day after sleeping in an oxygen-enriched atmosphere compared with sleeping in ambient air (5). Possible mechanisms include less fluid accumulation in the lung associated with acute mountain sickness, or a change in the contr

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https://doi.org/10.1007/978-3-662-31493-7 Patchy vasoconstriction may expose parts of the capillary bed to high pressure resulting in stress failure of the capillary wall. The development of an inflammatory process may then occur after the initiation of the leak.

Hallowed

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https://doi.org/10.1007/978-3-663-15751-9ts responsible for some of these unique properties have now been identified and are being characterized under physiological and disease conditions. These include the proteins involved in formation and assembly of tight junctions; the plasma membrane-embedded proteins that are responsible for transpo

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https://doi.org/10.1007/978-3-658-26763-6the release or action of a given autacoid. Among the mediator candidates considered, bradykinin is the only one to meet all criteria. Histamine, arachidonic acid and free radicals including nitric oxide may also be considered mediators of brain edema, but for each of these compounds evidence is less