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Titlebook: Hypothermia for Acute Brain Damage; Pathomechanism and P Nariyuki Hayashi (Professor),M. Ross Bullock (Prof Conference proceedings 2004 Spr

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Mathematical Analysis of Extremity Immersion Cooling for Brain Temperature Management relative humidity) until the brain temperature rises to 39°C, then continuing to rest for 1 h under one of the following treatments: (A) no cooling; (B) hands immersed in 10°C water; (C) feet immersed in 10°C water; (D) hands/feet immersed in 10°C water. The simulation results indicate that within
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Neuromyelitis Optica Spectrum Disorders, period. Following the hypothermie treatment, animals were rapidly (30 min) or slowly (90 min) rewarmed. Three days following TBI, animals were perfusion-fixed and quantitatively assessed for contusion volume, numbers of NeuN-positive cortical neurons, and axonal pathology. Post-traumatic hypothermi
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Mathematics and Its Applicationsvs 82.4% ± 0.1%; . < 0.01). The decrease of brain water content was accompanied with a significant reduction in BBB permeability to Evans blue dye (29.4 ± 5.3 vs 11.6 ± 3.0ng/g wet tissue; . < 0.05) and in accumulation of polymorphonuclear leukocytes (3.03 ± 0.79 vs 0.27 ± 0.16 units/g wet tissue; .
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J. W. ten Cate,M. H. J. van Oersdevices are tested against one another are necessary to further evaluate their usefulness in the intensive care unit setting. Also, the effect on final outcome of the different methods should be evaluated. Finally, future directions for the application of hypothermia in brain protection are suggeste
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