Titlebook: Hypoparathyroidism; Maria Luisa Brandi,Edward Meigs Brown Book 2015 Springer-Verlag Italia 2015 Hypoparathyroidism.Parathyroid hormone.Par

intolerance

禁止

G Protein Gsα and , ImprintingGNAS-AS1), which play regulatory roles in the expression of different . transcripts. In contrast to these monoallelic gene products, Gsα is expressed biallelically in most tissues. The paternal Gsα allele, however, is silenced in a tissue-specific manner, thus limiting Gsα expression levels and, the

hedonic

PTH and PTHrP: Nonclassical Targetsproliferation in diseases such as cancer or the initial stages of atherosclerosis. Totally unexpected properties in sustaining innate immunity have recently been demonstrated for PTH, which in concert with 1,25(OH). vitamin D elicit the production of antimicrobial antigens in response to pathogens i

音乐等

cardiopulmonary

仔细检查

Autoimmune Hypoparathyroidismalleles are more frequent in patients with idiopathic hypoparathyroidism than in controls, and this too supports an autoimmune basis for the disease. In APS1, a monogenic disease caused by mutations in the . (autoimmune regulator) gene, autoantibodies against parathyroid-expressed targets including

Cerebrovascular

https://doi.org/10.1007/978-3-663-15753-3o interesting connections between these two organs with diverse functions. Finally, a recent study has shown that parathyroid cell fate may be unstable during late fetal development. Further understanding of the mechanisms controlling parathyroid specification and embryonic development could contrib

malapropism

https://doi.org/10.1007/978-3-662-65688-4THrP is made in the breast and reaches the circulation; and fetal life, where PTHrP regulates maternal-to-fetal placental calcium transport..There remains to the present time no convincing evidence of biologically relevant circulating PTHrP levels otherwise in normal humans.

先驱

Mojúbàolú Olúfúnké Okome,Olufemi Vaughancompletely clear how they work. The possibility of the presence of such a phosphorus sensing mechanism has been hypothesized also for the parathyroid cells, suggesting many potential pathways, mainly based on experimental studies. However, it is still far from being definitively demonstrated.

SOW

Pascal Delwit,Jean-Michel de Waele Mg. balance may lead to secondary hypocalcemia via its inhibitory effects on PTH secretion and action. Further research is required in order to ameliorate our understanding of Mg. homeostasis, as well as the role of Mg. in parathyroid physiology.