Titlebook: Hyperthermic Oncology from Bench to Bedside; Satoshi Kokura,Toshikazu Yoshikawa,Takeo Ohnishi Book 2016 Springer Science+Business Media Si

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,Ein dynamisches Analyse-Modell für Raum,h causes intriguing biological responses such as tumor-specific immune response. These results suggest that MNHT is able to kill not only tumors exposed to heat treatment, but also unheated metastatic tumors at distant sites. Currently, some researchers have started clinical trials, suggesting that the time has come for clinical applications.

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Cell Cycle Responses to Hyperthermiaresulting in the delay of progression to the S-phase and cell cycle arrest at the G. and/or G./M phases of the cell cycle. Here, I summarize the cell cycle response provoked by HS with an aim to provide significant information for the potential of hyperthermia for use in cancer treatment.

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DAUNT

Enhancement of Hyperthermia on Anti-tumor Drug Sensitivityrough hyperthermia (Fig. .). In this chapter, the main focus is on the increase in activity of anti-tumor drugs induced by hyperthermia. DNA repair inhibition induced by hyperthermia has been described in other chapters.

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Satoshi Kokura,Toshikazu Yoshikawa,Takeo OhnishiBroadens understanding of the rudimentary mechanism of the anti-cancer effect of hyperthermia.Provides the latest clinical outcomes by hyperthermia treatment of cancer.Presents the future perspectives

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https://doi.org/10.1007/978-981-10-0719-4Anti-cancer; Chemotherapy; Epithelial-mesenchymal transition; Heat Shock Protein; Hyperthermic treatment