Titlebook: Human Trophoblast Neoplasms; Roland A. Pattillo,Robert O. Hussa Book 1984 The Editor(s) (if applicable) and The Author(s), under exclusive

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Epidemiology of Trophoblast Disease in Africa — Lagos in the past been linked with external factors including viral (4), chemical (5), dietary (6, 7), and genetic (8, 9). It has also been suggested (9) that trophoblastic neoplasms may be attributed to immune mechanisms.

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Genetic Origin, Immunobiology, and Gonadotropin Expression in Trophoblast and Nontrophoblast Neoplas 50:50 ratio of chromatin-positive to chromatin-negative moles. The initial large study by Baggish and Woodruff (1) on moles treated in Singapore revealed that all 90 moles studied showed well-defined heterochromatin bodies.

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Complement System in Abnormal Pregnancy and Chorionic Neoplasia: Immunotherapy and Chemotherapy of C. Is it possible that there is a compensatory mechanism for the suppressed cellular immunity? From the phylogenetic point of view, when cellular immunity is suppressed, the C. system of complement must be enhanced.

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Hallowed

Epidemiologic Aspects of Gestational Trophoblastic Diseases in Ibadan, Nigeriacidence (3). In the early and mid-sixties, Hendrickse in Ibadan, Nigeria (4), Oettle in Southern Africa (5), and Dodin (6) in Madagascar (now Malagasy) raised the possibility that similar lesions were encountered more frequently by physicians working in Africa than by their colleagues in Europe or North America.

Progesterone

Immunochemistry of Human Chorionic Gonadotropinobtained through immunoassay of the hormone or its subunits, we have chosen to focus this review on that background information which relates to immunochemical recognition of the hormone. Numerous other summaries are available dealing with the biological role of the hormone and its action at target organ receptors (3-5).

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JEER

Complete and Partial Hydatidiform Moles: Cytogenetic and Morphological Aspectsneation of at least two distinct molar syndromes: the complete, diploid, androgenetic mole of a 46,XX or 46,XY karyotype without an embryo/fetus; and the partial mole with a fetus and a 69,XXY, XXX, or XYY karyotype. Only the classic, complete mole retains at present a known association with chorionic cancer.

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