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Titlebook: Human Pathobiochemistry; From Clinical Studie Toshitaka Oohashi,Hirokazu Tsukahara,Fumio Otsuka Textbook 2019 Springer Nature Singapore Pte

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Acute Myeloid Leukemia: Mutations Blocking Differentiation Lead to Distinct Leukemic Subtypesormal ≤1.1) and prothrombin time of 17.1 s (normal 8.9–11.4 s) and a decreased fibrinogen of 106 mg/dL (normal 217–420 mg/dL). A peripheral blood smear presented numerous circulating immature myeloid cells intermediate in size with scant cytoplasm, azurophilic granules, and folded nuclei (Fig. 15.1)
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α1-Antitrypsin Deficiencytion tests results were normal. Serum protein electrophoresis failed to detect a peak corresponding to α1-globulin; subsequent determination of serum levels of AAT disclosed severely decreased concentrations (20 mg/dl; normal range, 170–274; measured by nephelometry, SRL, Tokyo, Japan). Arterial blo
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Hereditary Anticoagulant Deficiencieses are physiologically lower in childhood than in adulthood. Unexplained dissociation among PC, PS, and factor VII activity levels may portend a diagnosis of heritable PC or PS deficiency. The coagulation studies by using the age-dependent standards have then high sensitivity in screening for antico
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Learning including Small Group Discussion (SGD) and Problem-.This textbook uses a case-study approach to present the core principles of biochemistry and molecular biology in the context of human disease to students who will be involved in patient care. The 29 clinical cases have been carefully selec
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