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Titlebook: Herpes Simplex and Pseudorabies Viruses; A. S. Kaplan Book 1969 Springer-Verlag/Wien 1969 Herpes simplex-Virus.Pseudowutvirus

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Abortive Infection,al components are synthesized, no infectious virus is produced, this type of infection may be considered abortive. (This definition excludes nonsusceptible cells which do not adsorb virus.) An example of this kind of infection is provided by the interaction of the MP strain of herpes simplex virus w
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Pathogenesis,ections that will be described later. However, introduction of herpes simplex virus into susceptible animals results in severe infection. Herpes simplex virus can be transmitted experimentally to rabbits, guinea pigs, mice, rats, hamsters (., 1944), cotton rats (. and ., 1947), chick embryos (. and
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Pathology,cellular changes are the same in all infected tissues and have been described in detail in the section on Cytopathology. In addition to the cytological alterations common to all tissues, cytopathic changes will occur that are characteristic for certain tissues. Thus, in the skin, the mucous membrane
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Treatment,ath. These measures consist generally of transfusion of parenteral fluids, plasma and blood transfusions, plus symptomatic measures and good nursing care. In some cases, γ-globulin as a prophylactic measure for infants with eczema has been suggested (. and ., 1965). The prevention of acidosis and de
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Epidemiology,atients with frequently recurring herpes labialis all had antibody in their sera. Later studies by . et al. (1938) and . and . (1939) showed that children without neutralizing antibody developed stomatitis as a result of infection with herpes simplex virus; on recovery these children developed antib
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Latent and Persistent Virus,ars and enters into a quiescent, latent state. Infection recurs only as a reaction to certain stimuli. External factors such as irradiation or skin irradiation, as well as internal factors, such as certain pyrogenic-inducing diseases, will cause the development of facial herpes. The disease has also
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