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Titlebook: Heine-Jahrbuch 2021; Sabine Brenner-Wilczek Book 2021 Springer-Verlag GmbH Deutschland, ein Teil von Springer Nature 2021 Literaturgeschic

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ted, as a rule, not only to the initial structural injury to the brain, but also to a variety of secondary pathophysiological mechanisms, the common denominator of which is ischemia. The ischemic insult apparently initiates several known (and probably many others yet unknown) metabolic/biochemical c
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Jocelyne Kolbuch tissue injury. The selection of the small intestine as an example is made for two reasons: this organ represents those that early suffer from inadequate microcirculation in critical illness and, moreover, the consequences of gut tissue injury are thought to be of significant importance for the f
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Ortwin Lämke’s safety, as suggested by the Society of Critical Care Medicine [1–4]. However, only recently have technological developments resulted in the availability of capnography at the patient’s bedside, to measure variations in carbon dioxide concentration (CO.) and tidal volume simultaneously, during the
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Robert Steegerspontaneous breathing. These effects are secondary to an altered mechanical behaviour of the respiratory system. In addition, artificial ventilation is mostly undertaken in humans who are either anaesthetised or suffering from respiratory failure. Thus, most observations on the effects of artificial
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Heine-Jahrbuch 2021978-3-662-64170-5Series ISSN 0073-1692 Series E-ISSN 2628-5312
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