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Titlebook: Heat Shock Proteins and Cytoprotection; Atp-Deprived Mammali Alexander E. Kabakov,Vladimir L. Gabai Book 1997 Springer Science+Business Med

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https://doi.org/10.1007/978-1-4615-6007-4ATP; cell; chaperone; chemistry; gene expression; genes; medicine; mitochondria; neoplasm; protein; protein sy
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Heat Shock Proteins and Cytoprotection978-1-4615-6007-4Series ISSN 1431-0414
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1431-0414 Overview: 978-1-4613-7752-8978-1-4615-6007-4Series ISSN 1431-0414
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Molecular Biology Intelligence Unithttp://image.papertrans.cn/h/image/425004.jpg
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Alexander E. Kabakov Ph.D,Vladimir L. Gabai Ph.Dno length scale. The strengthening mechanisms at the basis of the reinforcing action exerted by nanoparticles (Orowan mechanism, enhanced dislocation density, grain refinement and load bearing effect) is described and their contribution discussed; modelling of the above mentioned mechanisms is also presented.
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ernehmen die Möglichkeit eines vereinfachten Zugangs zu den Organisationen im Stakeholder-Umfeld. Interessant ist das etwa für internationale Unternehmen, die fern des Heimatmarkts Zugang zu Verbänden und Vereinigungen in bestimmten Wirtschaftsräumen und Regionen suchen, um sich in der lokalen Geschäftswelt zu etablieren.
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ATP Homeostasis, Ionic Balance and Cell Viability,tion and (2) how can HSPs rescue ATP-depleted mammalian cells from death. To answer these questions it is necessary to first examine the mechanisms of ATP homeostasis and the effect of ATP depletion on some important cellular functions and viability.
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Involvement of Heat Shock Proteins in Protection of Various Normal and Tumor Cells from Ischemic Intransgenic animals (see the previous chapter). For other organs and tissues, adequate experimental evidence is absent. However, there is are numerous data indicating the possible involvement of HSPs in anti-ischemic protection. Below we discuss the main results obtained on this issue to date.
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