Titlebook: Heat Shock Proteins and Cytoprotection; Atp-Deprived Mammali Alexander E. Kabakov,Vladimir L. Gabai Book 1997 Springer Science+Business Med

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ATP Homeostasis, Ionic Balance and Cell Viability,tion and (2) how can HSPs rescue ATP-depleted mammalian cells from death. To answer these questions it is necessary to first examine the mechanisms of ATP homeostasis and the effect of ATP depletion on some important cellular functions and viability.

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ATP Depletion as Inducer of Heat Shock Protein Expression,ll eventually result in cellular death. However, transient energy deprivation is well-tolerated by cells; moreover, as in the case of heat shock, the cells subjected to a reversible ATP decrease become tolerant to subsequent and more prolonged energy starvation. This adaptive response may be governe

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Heat Shock Proteins and Cardioresistance to Ischemia,SPs help cells survive prolonged ATP deprivation. The answer is yes, such cell tolerance exists in reality and furthermore, it represents a special interest to the medical community because there are a number of pathological states which lead to long-term energy starvation in some human tissues and

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Involvement of Heat Shock Proteins in Protection of Various Normal and Tumor Cells from Ischemic Intransgenic animals (see the previous chapter). For other organs and tissues, adequate experimental evidence is absent. However, there is are numerous data indicating the possible involvement of HSPs in anti-ischemic protection. Below we discuss the main results obtained on this issue to date.

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e of Al toxicity and tolerance/resistance is fundamental to identify and improve crop productivity which is a better strategy than liming the soils as the latter is labor intensive, ineffective, and expensive. In this chapter, all aspects of Al toxicity and tolerance are discussed in a historic pers