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Localizing Expression of the Three Members of the Nerve Growth Factor Family to Neurons in the Brair “relatives,” forming a fascinating family of factors now known as the NGF family (see Ebendal et al. 1991). This new development began with the landmark report of Barde and colleagues (Leibrock et al. 1989), in which they showed that brain-derived neurotrophic factor (BDNF) had a remarkably high s

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Nerve Growth Factor: Studies Addressing Its Expression and Actions in the Central Nervous System,d significance of their actions it will be important to identify specific candidate molecules, link their expression and actions with the development of specific neuronal populations, and examine the effect of blocking their actions on neuronal development. The studies reported herein address the po

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Nerve Growth Factor Gene Expression in Actively Growing Brain Glia, on distant, projecting perikarya. To define the basis of local action, we used a sensitive nuclease protection assay to identify cells expressing the NGF gene in vivo and in vitro. In addition to neurons, glia from a variety of developing brain areas synthesized NGF mRNA, suggesting that CNS glia e

bourgeois

Human Nerve Growth Factor Receptor Expression in the CNS of Transgenic Mice, Alzheimer’s disease. Cholinergic cells of the basal forebrain express NGF receptors and carry out retrograde axonal transport of .I-NGF. We have generated transgenic mice that express human NGF receptors. Distinct neuronal expression in cholinergic cells of the basal forebrain and Purkinje cells of

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landfill

,Distribution and Characterization of β-Nerve Growth Factor Receptors in Alzheimer’s Disease, treatment of Alzheimer’s disease (AD). If β-NGF is used as a therapy, the presence of receptors on functioning neurones will be required. We have therefore examined the number and size distribution of neurones expressing β-NGF receptors in the nucleus of the diagonal band and the anterior, intermed

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Regulation of Nerve Growth Factor Synthesis in the Central Nervous System by Cytokines and Implicattage of the disease, and that the consequences of these degenerative changes are predominantly responsible for the impaired cognitive functions. Although there is no evidence that Alzheimer’s disease results from a reduced production of nerve growth factor (NGF), potential beneficial therapeutic eff

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Nerve Growth Factor and Behavioral Recovery After Brain Damage in Rats,ions. Since the late 1970s, evidence has accumulated indicating that nerve growth factor (NGF) acts as a neurotrophic factor for cholinergic neurons of the basal forebrain. There are conflicting data concerning the action of this protein on cognitive functions, particularly in animals with lesions a

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Functional Impairments that Develop in Nerve Growth Factor-Sensitive Neuronal Projections in the PNem, the collateral sprouting of undamaged nociceptive nerves into adjacent denervated skin. This new growth leads to an expansion of the surviving nociceptive fields. We now find that the . effect, a (reversible) ., particularly of the heat-nociceptive fields, is produced by the antibody-induced chr