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Titlebook: Glucagon III; Pierre J. Lefèbvre Book 1996 Springer-Verlag Berlin Heidelberg 1996 Diabetes.GLP-1.Glukagon.glucagon.molecular biology.patho

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Physiology and Pathophysiology of GLP-1,s — was compared (. et al. 1964; . and . 1967). A much higher insulin secretion was found after oral glucose. It was calculated that up to 50% of the insulin release after oral glucose was triggered by the “entero-insular axis,” a term introduced in the literature in 1969 (. and . 1969).
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Book 1996in medical imaging or in emergency medicine. Chapters are devoted to newly identified members of the glucagon family such as glucagon-like peptide-1 (GLP-1) and oxyntomodulin. .Glucagon III. is a comprehensive review of all information published on this important hormone since 1983 and is .the. reference book on the subject.
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Glucagonoma and Its Management,ported (. et al. 1974; . 1981; . and . 1981; . and . 1981; . et al. 1982) and particularly in Chap. 43 of the previous volume in this series, . (. et al. 1983). Since then, several reviews have been devoted to this syndrome (. et al. 1984; . 1985; . and . 1987; . et al. 1988; . and De . 1988; . 1989; . and . 1992).
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Book 1996cagon encyclopedia" and as such have no competitors in the scientific literature worldwide. In this volume, the most recent data on glucagon molecular biology are reviewed together with clinically relevant information on the role of glucagon in the pathophysiology of diabetes, the place of glucagon
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C Basics: Variables and Operators,g excessive glucagon secretion and/or action. Specific characteristics of the diabetic state associated with the glucagonoma syndrome are reviewed in more detail in Chap. 15 of this volume while the search for new glucagon antagonists is considered in Chap. 8.
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Glucagon and Diabetes,g excessive glucagon secretion and/or action. Specific characteristics of the diabetic state associated with the glucagonoma syndrome are reviewed in more detail in Chap. 15 of this volume while the search for new glucagon antagonists is considered in Chap. 8.
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