Titlebook: Glaucoma Update IV; G. K. Krieglstein (Direktor) Conference proceedings 1991 Springer-Verlag Berlin Heidelberg 1991 Elektrophysiologie.Gla

出处

gusher

Flirtatious

Estimable

https://doi.org/10.1007/978-1-4615-5573-5tly below 21 mmHg. Despite the above definition LTG does not always refer to a single clinical entity, but encompasses various etiologic disorders with unknown causes [7]. The pathogenesis of LTG is poorly understood, however, discrimination of cases may be possible from their clinical course; namel

ATRIA

Experimental Modulation of the Uveal Vasculature,ing the regulatory response of ocular vessels to physiologic and pharmacologic stimuli. For these studies, we have used a methacrylate intraluminal casting model using a modification of Batson’s compound #17 injected into major arterial vessels, the aorta or carotid arteries of experimental animals

ARK

The Response of the Meshwork Cells to Adrenergic Agents and Their Antagonists, of progress is surprising in view of the facts that nearly 100 years ago the major catecholamine component of the adrenal gland was isolated and named epinephrine [1], and that adrenal gland extracts were shown to lower IOP when injected subconjunctivally [2] or applied topically in other early exp

胆汁

In Vitro Correlates of Glucocorticoid Effects on Intraocular Pressure,s major inductions of specific proteins and glycoproteins which are not present in HTM cultures without glucocorticoid treatment. Evaluations of the DEX inductions in HTM cytosol and media fractions demonstrate a time course and dose-response consistent with clinical studies of steroid-induced eleva

助记

In Vitro Electrophysiology of the Ciliary Epithelium,ssure differences across the tissue. Ultrafiltration (dialysis under pressure) does not appear to be a major mechanism for aqueous humor production. Hydraulic conductivity and osmotic fluid permeability across the iris-ciliary body are too low to account for the formation of aqueous humor in rabbits

Injunction

The Ciliary Muscle and Nerves After Ciliary Ganglionectomy, et al. 1984). The ciliary muscle is parasympathetically dominated, and muscarinic cholinomimetic agonists have therefore been used to treat glaucoma for over a century (Bárány 1962). However, many aspects of ciliary muscle physiology and pathophysiology remain poorly understood and a parasympatheti