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Titlebook: Genomic Adaptability in Somatic Cell Specialization; Marie A. DiBerardino,Laurence D. Etkin Book 1989 Springer Science+Business Media New

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书目名称Genomic Adaptability in Somatic Cell Specialization
编辑Marie A. DiBerardino,Laurence D. Etkin
视频video
丛书名称Developmental Biology
图书封面Titlebook: Genomic Adaptability in Somatic Cell Specialization;  Marie A. DiBerardino,Laurence D. Etkin Book 1989 Springer Science+Business Media New
描述I am pleased to include this book as Volume 6 of Developmental Biology: A Comprehensive Synthesis. It has been edited by two of the foremost investiga­ tors in the study of genomic adaptability. lowe a special debt of gratitude to Dr. Marie A. DiBerardino, who developed the concept of the volume. Dr. DiBerar­ dino is also a very active member of the editorial board for this series. Much of the success of this series is due to her valuable advice. This series was established to create comprehensive treatises on specific topics in developmental biology. Such volumes serve a useful role in develop­ mental biology, since it is a very diverse field that receives contributions from a wide variety of disciplines. This series is a meeting ground for the various practitioners of this science, facilitating an integration of heterogeneous infor­ mation on specific topics. Each volume is intended to provide the conceptual basis for a comprehen­ sive understanding of its topic as well as analysis of the key experiments upon which that understanding is based. The specialist in any aspect of developmen­ tal biology should understand the experimental background of the field and be able to place th
出版日期Book 1989
关键词Embryo; Imprinting; development; developmental biology; genes; oogenesis; phenotype; tissue
版次1
doihttps://doi.org/10.1007/978-1-4615-6820-9
isbn_softcover978-1-4615-6822-3
isbn_ebook978-1-4615-6820-9
copyrightSpringer Science+Business Media New York 1989
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Making Global Partnership Work,changes and the circumstances and mechanisms associated with their occurrence. Stable changes in the structural and functional properties of cells are generally based on modifications at the level of DNA. Five kinds of modifications are known: (1) elimination of DNA sequences; (2) amplification of s
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https://doi.org/10.1007/978-3-319-64510-0ental products, each of these carries a spectrum of identifying markers: distinctive patterns of enzyme activities or unique metabolic products, diagnostic cytoarchitecture or elements of fine structure, and other specificities recognizable microscopically or biochemically. Phenotypic changes in ear
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,PDA at Work—Exploring Impact in Stability, patterns of protein synthesis. These unique constellations of proteins confer on cells the functional and structural characteristics that enable them to perform their specialized roles in the organism. However, in addition to the overt protein synthetic profile, cells may retain the potential to pr
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https://doi.org/10.1057/9780230118379elman and Kafatos (1974) to denote the change of the cuticular cells of the moth larval silk gland to those producing HCO. during metamorphosis/development and has since been used in many different contexts. So as not to produce a welter of semantics to replace the term already in use, I shall inste
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https://doi.org/10.1057/9781137397881 types acquire a specific cellular phenotype for performing a specialized function, no further changes usually occur. We call this the stability of the differentiated state. It is now widely accepted that the process and stability of cell specialization are under genetic control. But the frequency a
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https://doi.org/10.1057/9781137383501d. While some have concluded that cancer cells must arise by the dedifferentiation of normal cells, others consider it more likely that many cancer cells develop from normal cells that never fully differentiated. Since these undifferentiated cancer cells can be shown to share many fundamental charac
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978-1-4615-6822-3Springer Science+Business Media New York 1989
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