Titlebook: Genes, Oncogenes, and Hormones; Advances in Cellular Robert B. Dickson,Marc E. Lippman Book 1992 Springer Science+Business Media New York 1

Commonplace

Preamble

Left Ventricular Contactile Reserveating that the transduced oncogenes of certain acute transforming retroviruses represent activated versions of growth factors or growth factor receptors. When incorporated within the retroviral genome, such transduced sequences acquire the ability to induce neoplastic transformation. Analysis of vir

Adulate

Paul P. Carbone,Douglass C. Tormeyntly characterized c-.B3, these molecules form the type 1 family of tyrosine kinase growth factor receptors [., .]. The epidermal growth factor receptor and possibly the other two molecules are activated by binding of ligand to their extracellular domain, which is thought to induce a conformational

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ARENA

mydriatic

Antony J. O’sullivan,Robert D. Burgoyneof all ages, but it is primarily a postmenopausal disease. This appears to be because prolonged exposure of the gland to ovarian hormones is required, but the disease is usually associated with a long latency of 20 years or more [.]. Excessive proliferation, dedifferentiation, genetic mutability, an

Landlocked

M. Ebadi,R. F. Pfeiffer,L. C. Murrinormones estrogen and progesterone, and an increasingly long list of growth factors. Like their nonmalignant counterparts, the growth, proliferation, and metastatic potential of breast cancer cells are markedly influenced by these substances.

Infant

Jean-Guy Chabot,RÉMi Quirion,Gerard Morel research. Because most breast cancers are initially dependent upon estrogens for continued growth, much of this research has focused on the role of estrogen receptor (ER) in the control of gene expression and mitosis [.], and on its use as a marker for hormone responsiveness and prognosis [.]. In a

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978-1-4613-6552-5Springer Science+Business Media New York 1992

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