Titlebook: Gene Regulatory Sequences and Human Disease; Nadav Ahituv Book 2012 Springer Science+Business Media, LLC 2012 Systems Biology

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书目名称Gene Regulatory Sequences and Human Disease影响因子(影响力)




书目名称Gene Regulatory Sequences and Human Disease影响因子(影响力)学科排名




书目名称Gene Regulatory Sequences and Human Disease网络公开度




书目名称Gene Regulatory Sequences and Human Disease网络公开度学科排名




书目名称Gene Regulatory Sequences and Human Disease被引频次




书目名称Gene Regulatory Sequences and Human Disease被引频次学科排名




书目名称Gene Regulatory Sequences and Human Disease年度引用




书目名称Gene Regulatory Sequences and Human Disease年度引用学科排名




书目名称Gene Regulatory Sequences and Human Disease读者反馈




书目名称Gene Regulatory Sequences and Human Disease读者反馈学科排名

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Marina Metz,Olga Skriabina,Anna Samokhvalova can lead to human disease. In this chapter, we will define the different types of gene regulatory elements (promoters, enhancers, silencers, and insulators) and how to identify and functionally characterize them.

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gical advances that led to this breakthrough. In addition, several examples will be provided of nucleotide variants in noncoding sequences that have been shown to be associated with various human diseases.978-1-4899-8785-3978-1-4614-1683-8

iodides

Literatur der russischen Emigrant/innen cause dramatic changes to patterning, leading to congenital limb malformations. In this chapter, we will review characterized gene regulatory mutations leading to human limb malformations and also provide evidence that additional limb enhancers could be the cause of other human limb malformations.

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,-Regulatory Enhancer Mutations are a Cause of Human Limb Malformations, cause dramatic changes to patterning, leading to congenital limb malformations. In this chapter, we will review characterized gene regulatory mutations leading to human limb malformations and also provide evidence that additional limb enhancers could be the cause of other human limb malformations.

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Interkulturelle Medienkommunikationst a requirement for craniofacial-specific regulatory elements during . transcription in embryonic development and how the .-ruption of these elements alone might result in isolated PRS, an endophenotype of CD.