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Titlebook: Gene Regulation by Steroid Hormones III; Arun K. Roy,James H. Clark Conference proceedings 1987 Springer-Verlag New York Inc. 1987 Activat

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Masayuki Miyasaka,Kiyoshi TakatsuMarkaverich and Clark, 1979; Watson and Clark, 1980) and malignanttissues. Type I sites represent the classical estrogen receptor, which has a high affinity for estradiol (.. = 0.1 − 1.0 n.) and is present in target tissues in relatively low quantities. Nuclear type II sites appear to be a specific
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Srividya Kidambi,Theodore A. Kotchenmone interacts with a specific receptor. The second step is the tight binding of the steroid-receptor complex to chromatin, where it modulates the transcription of specific genes. We have recently observed, in the case of the progesterone receptor, the existence of a third step involving a hormone-d
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Cystic Fibrosis: Biology and Therapeutics, Fig. 1, 1,25(OH).D. is formed in the kidney according to the calcium and phosphorus needs of the organism (Haussler and McCain, 1977). Its main functions are the stimulation of intestinal calcium and phosphate absorption as well as bone remodeling. In addition to its mineral conservation effects in
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https://doi.org/10.1007/978-3-642-73526-4eans, 1974; Liao, 1975; Muldoon, 1980; Schrader, et al., 1981; Schmidt and Litwack, 1982; Jensen et al., 1982; Ringold, 1985). Specific receptors for a number of steroid hormones have been purified (O’Malley and Schrader, 1976; Govindan, 1979; Kuhn et al., 1975; Pike et al., 1982; Jensen et al., 198
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Chronic Myelomonocytic Leukemia (CMML), proteins that are traditionally thought of as “extracellular” steroid transport proteins. Both proteins have been purified to apparent homogeneity, specific antibodies have been raised against them, and their physicochemical characteristics and physiological functions have been the subject of numer
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