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Titlebook: Gene Expression in Muscle; Richard C. Strohman,Stewart Wolf Book 1985 The Editor(s) (if applicable) and The Author(s), under exclusive lic

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Induction of Incoordinate Synthesis of Muscle Proteins by the Tumor Promoter TPA and the Carcinogen lag phase of hours between the earliest detection of these muscle-specific isoforms in mononucleated postmitotic myoblasts and their assembly into myofibrils. During this lag period, myofibrillar proteins and desmin appear to be widely distributed throughout the sarcoplasm.
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Book 1985nked genetic disorder. Up to the present, however, the offending gene has not been identified although its location on the short arm of the X chromosome has been approximately determined. The gene product associated with the initial disturbance in skeletal muscle has also remained elusive up to now.
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Wertewandel und Interessenartikulation,le involvement in the mechanisms of Duchenne dystrophy. The proteins of the myofibrils, myosins, actins and troponin have been and still are primary targets for study as they reflect the two distinct stages of muscle cell development, differentiation and maturation.
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the effects of cross innervation of a fast muscle with a slow motoneuron can be mimicked by chronic stimulation of the fast muscle’s own intact motoneurons at 5–10 Hz, the frequency of activity of a slow motoneuron (373, 374).
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