Titlebook: Gastric Cardiac Cancer; Qin Huang Book 2018 Springer International Publishing AG, part of Springer Nature 2018 Esophagogastric Junction.Ca

补助

Adjourn

https://doi.org/10.1007/978-3-322-92388-2presence in the gastric cardia in the East Asian population, compared to the Western Caucasian population; (3) cardiac mucosa is plastic and dynamic in response to changes in local microenvironment that modify its length and distribution patterns.

心胸狭窄

Aspekte einer Theorie des Romans, of reduction of the tumor stage. Molecular precision target therapy on HER2, PD-1, and PD-L1 molecules has been shown to be highly promising in treatment of this cancer with improved outcomes. There is an urgent need for randomized, multicenter, well-controlled studies to establish universally accepted optimal therapy guidelines for this cancer.

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LINE

Chemical Therapy, of reduction of the tumor stage. Molecular precision target therapy on HER2, PD-1, and PD-L1 molecules has been shown to be highly promising in treatment of this cancer with improved outcomes. There is an urgent need for randomized, multicenter, well-controlled studies to establish universally accepted optimal therapy guidelines for this cancer.

小口啜饮

The Esophagogastric Junction,lthough a universally accepted criterion for EGJ has not been established, the most commonly used landmark for endoscopic mucosal EGJ is the proximal end of gastric longitudinal mucosal folds with a high reproducibility in different populations. As to histologic EGJ markers, the distal ends of deep

Pigeon

Cardiac Mucosa,ts in various ethnic populations has firmly established the congenital nature of this mucosa with a few new features: (1) the length of cardiac mucosa is much shorter than previously believed; (2) the distrisbution pattern of cardiac mucosa varies among different populations with a more predominant

athlete’s-foot

野蛮

Natural History,n, gastric cardiac carcinoma is a slow-progressive malignant neoplasm, taking 2–6 years to progress from intramucosa to submucosa. However, once invading into the muscularis propria, the tumor becomes much more aggressive. Therefore, the patient management may need to be individualized, according to

PANEL

Clinical Molecular Pathology,mon molecular type is the chromosomal instable carcinoma that shows an increasing gradient from a low prevalence in the distal stomach to a high level in the gastric cardia and to the peak in the distal esophagus. Thus, esophageal adenocarcinoma is in fact part of gastric chromosomal instable carcin