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Titlebook: GI Microbiota and Regulation of the Immune System; Gary B. Huffnagle,Mairi C. Noverr Book 2008 Springer-Verlag New York 2008 antibiotics.b

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发表于 2025-3-21 20:01:57 | 显示全部楼层 |阅读模式
书目名称GI Microbiota and Regulation of the Immune System
编辑Gary B. Huffnagle,Mairi C. Noverr
视频video
图书封面Titlebook: GI Microbiota and Regulation of the Immune System;  Gary B. Huffnagle,Mairi C. Noverr Book 2008 Springer-Verlag New York 2008 antibiotics.b
出版日期Book 2008
关键词antibiotics; bacteria; diseases; immune response; immunity; immunology; infection; infections; infectious di
版次1
doihttps://doi.org/10.1007/978-0-387-09550-9
isbn_softcover978-1-4899-8816-4
isbn_ebook978-0-387-09550-9
copyrightSpringer-Verlag New York 2008
The information of publication is updating

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The Commensal Microbiology of the Gastrointestinal Tract,lenge. Microscopic observations have revealed that the majority of bacteria present in the GI tract are not detected using standard culturing techniques, however with the application of culture-independent techniques it has been estimated that between 500 to 1000 bacterial species inhabit the human
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Overview of the Gastrointestinal Microbiota,ts the combined effects of selection pressure on the part of the host and on the part of the microbes themselves. Through recent advances in the field of microbial ecology we are beginning to understand the forces that shape this complex community. We will review what is known about the interaction
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Diet, Immunity and Functional Foods, foods as a form of preventive medicine has been the subject of much research over the last two decades. It is well known that nutrition plays a vital role in chronic diseases, but it is only recently that data relating to the effects of specific nutrients or foods on the immune system have become a
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Host-Microbe Communication within the GI Tract,lth, while at the same time maintaining itself as an isolated environment to prevent infection and systemic disease. To maintain homeostasis in the gut, communication between the host and residing microbial communities must occur to identify and eliminate potential pathogens which could colonize and
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,The “Microflora Hypothesis” of Allergic Disease,al factors have important phenotypic consequences. Over a span of decades, a dramatic increase in the prevalence of allergic disease in westernized populations suggests the occurrence of critical changes in environmental pressures. Recently, it has been shown that the microbiota (i.e. microflora) of
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