Titlebook: GABA: Receptors, Transporters and Metabolism; Chikako Tanaka (Professor Emeritus, Director),Norm Book 1996 Birkhäuser Verlag 1996 Calcium.

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978-3-0348-9858-4Birkhäuser Verlag 1996

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https://doi.org/10.1007/978-3-0348-8990-2Calcium; GABA; Glutamat; Nervous System; biology; central nervous system; development; immunity; metabolism;

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Establishment of GABA as an Inhibitory Neurotransmitter at Crustacean Neuromuscular Junction and in 63 that inhibitory axons of lobsters contain about 200 times higher concentration of GABA than excitatory axons, which strongly suggested that GABA is an inhibitory transmitter. When I joined the Kuffler’s group in 1964, one crucial piece of evidence was missing, namely the demonstration of the rele

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Membrane Associated L-Glutamate Decarboxylase and Insulin-Dependent Diabetes Mellitus (IDDM)ified from porcine brain. M-GADI and M-GADII appear to be integral membrane proteins whereas M-GADIII is a peripheral protein attached to membranes in a Ca. dependent manner. In addition, M-GADI and M-GADII were found to be the major autoantigens in insulin-dependent diabetes mellitus (IDDM), also k

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Anti-GAD Antibody in Various Neurological Diseasesiabetes mellitus (IDDM) or other diseases and 461 normal subjects. Anti-GAD antibody was detected in the CSF of a patient with epilepsy and cerebellar atrophy who was administered phenytoin for more than 20 years. Anti-GAD antibody was detected in the serum of 36.6% patients with IDDM.