Titlebook: endoCANNABINOIDS; Actions at Non-CB1/C Mary E. Abood,Roger G Sorensen,Nephi Stella Book 2013 Springer Science+Business Media New York 2013

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https://doi.org/10.1007/978-1-4757-5876-4aims to summarise the work that became the first study to demonstrate a non-neuronal physiological role for GPR55 in vivo. In summary, male mice lacking GPR55 develop a high bone mass phenotype due to impairment in osteoclast function—consistent with GPR55 agonists O-1602 and LPI stimulating osteocl

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https://doi.org/10.1007/978-3-642-84850-6odegenerative effects in various experimental models. Here we review the interaction of cannabinoids (plant, endogenous or synthetic) with microglia, which are considered to be the immune cells of the brain. We describe the functional endocannabinoid system (ligands, receptors, and enzymes) in micro

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https://doi.org/10.1007/978-1-4684-7284-4can mediate fast inhibitory synaptic transmission, the 5-HT. receptors are involved in both excitatory and inhibitory synaptic transmission in the central and peripheral nervous system. These receptors play important roles in several physiological and pathological processes such as vomiting reflex,

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https://doi.org/10.1007/978-981-10-7371-7gene transcription. There are three peroxisome proliferator-activated receptor (PPAR) isoforms; α, δ (also known as β), and γ, which are activated by a numerous ligands including endocannabinoids. Activation of these receptors has been shown to modulate inflammation, in a number of different animal

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https://doi.org/10.1007/978-0-387-39975-1ome proliferator-activated alpha nuclear receptor (PPARα), which regulates genes involved in lipid metabolism and inflammatory responses, is a viable target for treating nicotine dependence. The endogenous ligands for PPARα, oleoylethanolamide and palmitoylethanolamide, are structurally similar to a

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