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Titlebook: Experimental and Clinical Neuropathology; Proceedings of the F Kurt Jellinger (Director),Filippo Gullotta,Mirosla Conference proceedings 19

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Ionic Shifts in Myelinated Nerve Fibers During Early Stages of Wallerian Degeneration nerve 18 h and 36 h after crush and in control nerves. The investigations were performed in freeze-dried ultrathin cryosections after shock freezing of the nerves in liquified propane. 18 h after crush, no definite alteration of the ions were found compared to the control nerves. In electron microg
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Preferential Vulnerability of Dendrites to Lithium Ion in Rat Brain and in Nerve Cell Culturese of 6–18 mmol/kg for 1–6 days. The most severe fine structural changes were revealed in the hippocampus. Low-dose (0.33 mmol/kg) lithium treatment lasting for a year did not cause any morphologically detectable alterations in the rat brain. In vitro studies showed a reduction of den-dro-axonal pro
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Effect of Prolonged Acetylcholinesterase Inhibition on Postnatal Brain Development in Rabbit I. Leveys starting on the 6th day of life or as a single dose on the 16th day of life. Animals were sacrificed 30 min, 3 und 24 h after treatment. The spinal cord and the brain were removed and 6 regions (neocortex, hippocampus, basal ganglia, mesencephalon, brain stem, cerebellum) were isolated. Acethylch
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The Activity of Some Hydrolytic Enzymes in the Brain After Administration of “Cynkotox”.0 g of Cynkotox was given daily to adult Wistar rats over a period of 10 days (the per os DL. for rats is 5 200 mg/kg of body weight)..The results have shown that chronic ingestion of large doses of Cynkotox may cause degenerative changes of neurons in various brain regions as well as alterations i
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Unusual Calcifications in a Monkey’s Brainr distributed in the ground substance of the nervous tissue. Laser spectrography analysis disclosed accumulation of calcium, phosphorus, silicium, magnesium and copper. No iron cations were found in the deposits.
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Influence of Damage of Hypothalamo-Hypophysial Neurosecretory System and Adrenalectomy on the Blood-ascular response to cerebral shock. The breakdown of this threshold could occur at the last stage of intracranial hypertension, being expressed by insufficient control of cardiovascular system with the fall of blood pressure and paralysis of cerebral blood vessels, BBB damage, that eventually might lead to brain death.
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