Titlebook: Experimental Hepatocarcinogenesis; M. B. Roberfroid,V. Préat Conference proceedings 1988 Plenum Press, New York 1988 DNA.apoptosis.biology

凹室

憎恶

persistence

The Induction of Localized Tumours by Carcinogens Implanted into the Livercute liver cell damage and necrosis and therefore would minimize or obviate the subsequent connective tissue proliferation. With luck, it was reasoned, the presence of a persisting depot of carcinogenic material would in time also lead to the development of tumours, as had been shown to be the case in other organs.

幻想

安装

Catherine W. Ernst,A. Marcos Ramosngle step protocols do exist for the chemical induction of cancer using large doses of carcinogen, most of the protocols which are classically used for that purpose are multisteps in that sense that they involve a sequence of different treatments.

Distribution

https://doi.org/10.1007/978-1-0716-2237-7ytes. These changes are thought to reflect an adaptive program in the liver that is switched on consequently to the increased functional demands on the organ (Conney et al. 1960; Argyris and Magnus 1968; Schlicht et al. 1967, 1968; Koransky et al. 1969; Schulte-Hermann 1974; Schulte-Hermann et al. 1980a,b).

消散

容易做

FLAG

out 100 scientists, mostly from Europe but also from the United States, met there for three days in a very friendly atmosphere to exchange knowledge and ideas on experimental and human liver carcinogenesis. The main topics discussed during the meeting included general reviews on hepatocarcinogenesis

存在主义

Genome Instability in Cancer Development different chemical nature. In experimental animals, mostly the rat and the mouse, quite a number of diverse chemicals have been demonstrated to produce liver tumors. Accordingly, such chemicals are called hepatocarcinogens.