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Titlebook: Experimental Hepatocarcinogenesis; M. B. Roberfroid,V. Préat Conference proceedings 1988 Plenum Press, New York 1988 DNA.apoptosis.biology

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书目名称Experimental Hepatocarcinogenesis
编辑M. B. Roberfroid,V. Préat
视频video
图书封面Titlebook: Experimental Hepatocarcinogenesis;  M. B. Roberfroid,V. Préat Conference proceedings 1988 Plenum Press, New York 1988 DNA.apoptosis.biology
描述The meeting on experimental hepatocarcinogenesis which took place in Spa, Belgium at the end of May 1987 was the Second European Meeting. About 100 scientists, mostly from Europe but also from the United States, met there for three days in a very friendly atmosphere to exchange knowledge and ideas on experimental and human liver carcinogenesis. The main topics discussed during the meeting included general reviews on hepatocarcinogenesis, experimental models of hepa­ tocarcinogenesis, biology of hepatocarcinogenesis, and in vitro studies in hepatocarcinogenesis. They are all covered by the various chapters of this proceedings volume, which reflects the present state of knowledge in this important field of cancer research. The final aim of that research is to understand the basic mechanisms of carcinogenesis. The liver offers a parti­ cularly interesting tool to reach such a goal. Indeed, its biochemistry, its morphology, and its physiology are very diverse, but relatively well known. Various protocols have been developed to produce hepatocellular carcinomas or other malignant tumors. Their appearance is most often preceded by phenotypically altered foci and nodules which have been i
出版日期Conference proceedings 1988
关键词DNA; apoptosis; biology; cancer; carcinogenesis; carcinoma; cell; cell death; cellular differentiation; chemi
版次1
doihttps://doi.org/10.1007/978-1-4613-0957-4
isbn_softcover978-1-4612-8264-8
isbn_ebook978-1-4613-0957-4
copyrightPlenum Press, New York 1988
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Genome Mapping and Genomics in Animals, the carcinogenic process can either evolve naturally or operationally be modulated. The factors that modulate an initiated carcinogenesis can have positive or negative effects. When the incidence and/ or the yield of cancer is increased and/or the latency period is decreased by the modulating trea
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Genome Mapping and Genomics in Animalsty (2), enzymatic competence of tissue(s) to activate the procarcinogen (3–5), the interaction of ultimate carcinogenic moiety with specific macromolecular site(s) (6,7), the rate of removal of the formed adduct(s) (8), the degree of macromolecular repair (9,10), caloric intake (11–13), immune compe
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Katherine M. Buckley,Jonathan P. Rastd with PHC are different in low incidence areas of the world such as Europe and U.S.A. /Friedman, 1983; Lapis and Johannessen, 1979; Sandler et al., 1983; Schaff et al., 1971/ and in high incidence areas, such as sub-Saharan Africa /Anthony et al., 1973; Kew, 1978; Szmuness, 1978/, Southeast Asia /C
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NATO Science Partnership Subseries: 3ation, localization and quantification of specific substances, reactive groups and sites of enzymatic activities in tissues, cells and cell organelles. Principally metabolic products or enzymes can also be assessed by biochemical analysis nowadays much more efficiently because of the development of
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