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Titlebook: Experimental Hematology Today—1985; Selected Papers from S. J. Baum,Dov H. Pluznik,L. A. Rozenszajn Conference proceedings 1986 Springer Sc

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Glycosphingolipids as Specific Differentiation-Markers and Differentiation-Inducers for Human Myeloghis phenomenon gives profound therapeutic importance as well as one of the basic experimental models, providing a clue for elucidating the fundamental mechanisms of regulation for growth and differentiation of hematopoietic cells.
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Immunoglobulin and T-Cell Receptor Gene Rearrangements in Human Acute Leukemiase overcome by utilizing the DNA rearrangement of immunoglobulin (Ig) and T cell receptor genes to reveal the clonality, cellular lineage, and stage of differentiation of such acute undifferentiated leukemias (AUL) (2).
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https://doi.org/10.1007/978-3-642-71505-1ctivity [3]. We report here results of studies designed to identify a method of concentrating human serum without loss of its biologically active Ep content, and when unsuccessful in this endeavour, studies were initiated to determine why Ep activity is invariably lost after concentrating human sera.
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https://doi.org/10.1007/978-3-658-30690-8believed to be targets of acute GVHD. Despite considerable data in animal models and in humans, the precise role of other effector mechanisms such as humoral immunity, antibody-dependent cellular cytotoxicity, natural killer cells, lymphokines, and the monocyte-macrophage system, remain controversial.
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https://doi.org/10.1007/978-3-662-58643-3ial Seattle experience with autologous marrow transplantation as treatment for patients with relapsed or resistant malignant lymphoma and compares this approach with our previously published experience using syngeneic and allogeneic transplantation in a similar group of patients (3–5).
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Dependence of CFU-S Proliferation on the CFU-S Populationd differentiation resulted in migration of CFU-S from the marrow to spleen (13) and in both organs their proliferation was inversely proportional to their concentration. These local variations in proliferative activity of the CFU-S population suggest that there is local control over CFU-S proliferation.
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