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Titlebook: Excitotoxins; Kjell Fuxe,Peter Roberts,Robert Schwarcz Book 1983 Palgrave Macmillan, a division of Macmillan Publishers Limited 1983 biolo

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楼主: 门牙
发表于 2025-3-30 09:19:37 | 显示全部楼层
Mechanisms Of Excitotoxins Examined In Organotypic Cultures Of Rat Central Nervous System, 1980), kainic acid (KA), produce axon-sparing neurodegenerative lesions in the striatum of the rat. Ablation of corticostriatal fibers before such injections abolished the neurotoxic effects of KA (McGeer et al, 1978; Biziere and Coyle, 1978). The consequences of KA administration have now been wi
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Mechanisms Of Excitotoxins Examined In Organotypic Cultures Of Rat Central Nervous Systemle et al, 1977; Fibiger, 1978). Our studies (Whetsell et al, 1979; Whetsell, 1979; Whetsell and Nicklas, 1980) in organotypic cultures of rat striatum (CA), striatum-plus-frontal cortex (CA-CX) and frontal cortex alone (CX), have demonstrated that the neurotoxic effects of KA can be observed only in CA-CX and CX cultures.
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Noor Kamal Al-Qazzaz,Alaa A. Aldoori tasks in the brain. It is involved in energy metabolism and fatty acid synthesis, incorporated into proteins and peptides and is even a precursor for the inhibitory neurotransmitter GABA. In addition its action as a neuroexcitant has been regarded as universal and non-specific.
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Jutta Wollersheim,Isabell Welpeental conditions the distant damage is largely due to the spread of paroxysmal discharge and not to a direct action of the toxin (by diffusion). The usefulness of KA to study the relationship between epilepsy and brain pathology is reviewed and the possible causes of the particular vulnerability of the ammon’s horn to epilepsy is discussed.
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