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Titlebook: Essential Nutrients in Carcinogenesis; Lionel A. Poirier,Paul M. Newberne,Michael W. Pari Book 1986 The Editor(s) (if applicable) and The

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Modification of Experimental Colon Carcinogenesis by Dietary Fibers,asingly apparent that the effect of dietary fibers on colon carcinogenesis depends on many factors, including the type and amount of fiber; the other dietary components, particularly fat; animal species, strain, and sex; and the type of carcinogen and its dose and route of administration. Despite su
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Dietary Cholesterol, Serum Cholesterol, and Colon Cancer: A Review,nesis. A mechanism for this possible relationship is currently under investigation. Additional international epidemiologic data, although not uniformly consistent, indicate an inverse relationship between serum or plasma cholesterol levels and risk for colon cancer. This risk is greatest at serum ch
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Dietary Protein and Experimental Carcinogenesis,he definitive studies concerning protein and cancer have utilized protein underfeeding and feed restriction. Optimal or less than optimal protein intakes have generally inhibited spontaneous and chemically induced tumor growth as well as the growth of transplantable tumors. Studies have focused on t
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Role of Tryptophan in Carcinogenesis,hemical carcinogens. For many years, tryptophan has been implicated in carcinogenesis of the bladder. The evidence regarding tryptophan’s effect on hepatic tumorigenesis is conflicting; an enhancing effect has been reported by some investigators, but a reduction in tumorigenesis has been reported by
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Lipotropic Factors and Oncogenesis, the past 6 decades. A thin thread of common interest in 1-carbon metabolism and a small band of dedicated individuals have kept this area of biology alive. Today, the lipotropes are enjoying a resurgence of interest and effort with promise for significant contributions to some of our most serious c
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Choline Deficiency and Chemical Carcinogenesis,arcinogenesis in experimental animals and discussed its possible mechanisms. Choline deficiency produces various pathologic lesions, involving virtually every organ of the body, as a result of a decrease in phospholipid and acetylcholine synthesis and in the supply of labile methyl groups. The liver
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