Titlebook: Epstein-Barr Virus and Human Disease; P. H. Levine,D. V. Ablashi,R. Glaser Book 1987 The Humana Press Inc. 1987 AIDS.Antigen.blood.disease

macrophage

surmount

S. Coppola,S. Froio,D. Chiumello(2), relative risks of NPC for the phenotypic expression of these antigens were estimated as 1.5 for A2, 2.14 for B17 and 1.88 for BW46. These associations suggest that either the presence of these HLA genes themselves or the occurrence of a non-HLA disease susceptibility gene (DSG) closely linked t

束缚

https://doi.org/10.1007/978-3-319-27349-5y pattern that is predictive of treatment effectiveness would be very useful for monitoring patients. Analyses of serial antibody titers are often complicated by censorship of the time to response. All patients will not experience the response of interest, e.g., death or disease recurrence, before t

Blood-Vessels

E. De Waele,P. M. Honore,H. D. Spapen decreased in general after radiation therapy(4,5). On the other hand, antibodies against H. simplex virus(HSV) and H. zoster virus (HZV) in NPC patients may be normal(6–8) or increased(9). The present communication revealed that the decrease of EBV-associated antibodies after radiation therapy was

occurrence

诗集

F. Guarracino,P. Bertini,M. R. Pinskyintimate contacts of an index case (1). Yet EBV infections must spread efficiently because most children become infected early in life (2). The present study addressed inconsistencies on transmission patterns of EBV in prior reports by prospectively evaluating families with a childhood index case of

phase-2-enzyme

P. Yang,A. M. Esper,G. S. Martinnd sleep disturbances of several years duration has been associated with consistently elevated antibodies to the early antigen of EBV.. High titers to early antigen (≥ 80) are suggestive of ongoing virus replication, since they occur during acute infectious mononucleosis reactivated disease in immun

Myosin

S. Aldekhyl,H. Tlayjeh,Y. Arabihoma, carcinomas originating in Waldeyer’s ring, thymus and B cell lymphomas in immunosuppressed patients. While IM is a polyclonal B cell proliferative disease in organ transplant recipients, polyclonal oligoclonal or monoclonal lymphoproliferative lesions carrying EBV genome can occur. Congenitall

VOC

L. Weaver,D. G. Bates,L. Camporotaere analyzed for EBV reactivities. Patients belonging to the first group survived more than seven years after the first diagnosis of the tumor, whereas in the second group patients died within one or two years.

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