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Titlebook: Epstein-Barr Virus and Human Disease; P. H. Levine,D. V. Ablashi,R. Glaser Book 1987 The Humana Press Inc. 1987 AIDS.Antigen.blood.disease

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书目名称Epstein-Barr Virus and Human Disease
编辑P. H. Levine,D. V. Ablashi,R. Glaser
视频video
丛书名称Experimental Biology and Medicine
图书封面Titlebook: Epstein-Barr Virus and Human Disease;  P. H. Levine,D. V. Ablashi,R. Glaser Book 1987 The Humana Press Inc. 1987 AIDS.Antigen.blood.disease
描述Since its discovery as the cause of infectious mononucleosis in 1964, the Epstein-Barr virus (EBV) has been etiologically implicated in an increasing number of human diseases. Generally considered the first human oncogenic virus because of a number of studies linking it with Burkitt‘s lymphoma and nasopharyngeal carcinoma (NPC), as well as its documented oncogenicity in nonhuman primates, EBVhas served as a model for identifying subsequent candidate oncogenic viruses and the stimulus for Evans‘ revision of the Henle-Koch postulates to accommodate the problems in proving viral oncogenicity in humans. Research on the role of EBV in human cancer was particularly en­ hanced (a) by the pioneering work of Werner and Gertrude Henle, and (b) by the coordinated efforts of the Special Virus Leukemia Program and its successors, the Special Virus Cancer Program and the Virus Cancer Program of the National Cancer Institute (NCI). Initiated by Dr. Frank J. Rauscher, who subsequently became director of the Ncr and is now Vice-President of the American Cancer Society, and expanded by Dr. John B. Moloney, whose contributions to cancer research were honored at this Second International Symposium on
出版日期Book 1987
关键词AIDS; Antigen; blood; diseases; infection; infections; interferon; leukemia; lymphocytes; phenotype; proteins;
版次1
doihttps://doi.org/10.1007/978-1-4612-4590-2
isbn_softcover978-1-4612-8940-1
isbn_ebook978-1-4612-4590-2
copyrightThe Humana Press Inc. 1987
The information of publication is updating

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Epstein-Barr Virus-Associated Lymphoid and Epithelial Lesions Occurring in the Life Cycle (BL), nasopharyngeal carcinoma (NPC), and infectious mononucleosis (IM) occurring in tropical Africa in regions with holoendemic malaria, Southeast China, and in western countries, respectively (1). Also, during this decade the immunobiology of EBV-induced diseases was elaborated.
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Anti-EBV Titers and the Application of a Prognostic Score to North American Patients with NasopharynNPC) (1–3), was conducted at the Mayo Clinic (Rochester, Minnesota) and at participating centers; it began in 1978. Detailed information was collected on each patient. Tumor stage was determined on the basis of the clinical data and was classified according to several systems (4–6). Blood for the an
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Nasopharyngeal Carcinoma in Yugoslavia: Geographical Distribution, Clinical, Pathological and Viroloeach having different life styles and food tradition.. In order to investigate the prevalence of nasopharyngeal carcinoma (NPC) in these different regions, a retrospective analysis of ENT tumors seen between 1981 and 1983 in eight different clinics was carried out. As seen in table 1, a ten-fold dif
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A Preliminary Analysis of HLA Studies on Multiple NPC Cases among Siblings from the People’s Republi(2), relative risks of NPC for the phenotypic expression of these antigens were estimated as 1.5 for A2, 2.14 for B17 and 1.88 for BW46. These associations suggest that either the presence of these HLA genes themselves or the occurrence of a non-HLA disease susceptibility gene (DSG) closely linked t
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EBV-Associated Antibodies and Other Antibodies in Nasopharyngeal Cancer Patients before and after Ra decreased in general after radiation therapy(4,5). On the other hand, antibodies against H. simplex virus(HSV) and H. zoster virus (HZV) in NPC patients may be normal(6–8) or increased(9). The present communication revealed that the decrease of EBV-associated antibodies after radiation therapy was
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Epstein-Barr Virus Infection in Families with a Childhood Index Case of Infectious Mononucleosisintimate contacts of an index case (1). Yet EBV infections must spread efficiently because most children become infected early in life (2). The present study addressed inconsistencies on transmission patterns of EBV in prior reports by prospectively evaluating families with a childhood index case of
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