Titlebook: Epigenetics of Infectious Diseases; Walter Doerfler,Josep Casadesús Book 2017 Springer International Publishing AG 2017 bacterial infectio

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Christoph Wittmann,Judith Beckerbacteria. One powerful strategy used by intracellular pathogens (e.g., .) is the secretion of nucleomodulins that manipulate chromatin structure in the host nucleus. The effects of this dialog are often limited in time, causing transient gene expression changes. However, increasing evidence suggests

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Toward Sustainable Amino Acid Production,r the virulence of a different range of bacterial pathogens of DNA methyltransferases other than Dam has been identified. This leads to new understanding of possible origins and occurrence of heterogeneity and virulent phenotypes in clonal populations. The contribution of new technologies to explore

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https://doi.org/10.1007/978-1-4684-3174-2ionships between white–opaque switching and pathogenesis, sexual reproduction, and other biological processes will be discussed. The newly discovered white–gray–opaque tristable switching system and white–opaque switching in two .-related species, . and ., will be included in the last two sections.

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Hans Brückner,Robert Wittner,Herbert Godel to the host epigenomic landscape. Changes in host DNA methylation, histone posttranslational modifications, nucleosome positioning, chromatin assembly, and regulation of transcription have been noted in the parasitized host. To date, only a few parasite-derived effectors have been shown to directly

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,Epigenetic Consequences of Epstein–Barr Virus Infection,sociated with latency is accompanied by repressive chromatin modifications and DNA methylation. Latency is integral to the lifelong persistence of EBV, yet persistence also relies on viral replication and virus production for infection of naïve hosts. Thus, EBV can overcome and disrupt the repressiv