Titlebook: Endothelin Receptors and Signaling Mechanisms; David M. Pollock,Robert F. Highsmith Book 1998 Springer-Verlag Berlin Heidelberg 1998 Activ

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https://doi.org/10.1007/978-3-658-18486-5r proteins 1 and 2 elements, shear stress response element, cAMP response element, among others) which mediate eNOS expression in response to shear stress, cyclic strain, transforming growth factor-β, basic fibroblast growth factor, or lysophosphotidylcholine. Various agonists elevating cytosolic ca

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Eberhard Abele,Philipp Kuske,Horst Langonary hypertension, ischemia reperfusion injury and organ infarctions, congestive heart failure, various forms of shock, and hypercholesterolaemia and atherosclerosis (see refs. 4, 5 for review). Clearly such increases can be secondary rather than causative to the pathological processes. However, th

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Jörg Fegert,Michael Kölch,Andrea Kliemanncle cells.. While numerous reports have described these actions of ET-1, the signaling mechanisms in these respective cell types are incompletely resolved. Some common themes involve receptor activation and mobilization of calcium as well as recruitment of a variety of second messengers. The focus o

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https://doi.org/10.1007/978-3-642-57938-7mitogenic response to growth factors. To understand ET-induced mitogenesis it is critical to understand how G-protein-coupled receptors activate this kinase cascade since it appears that this cascade transduces signals from cell surface receptors to the nucleus, thus altering the transcription of ge

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Introduction to the Endothelin System, filter allowing nutrients from the blood stream to diffuse through to the underlying tissues without letting proteins or blood cells escape. We now know that endothelial cells are important regulators of circulatory function, due in large measure to their recognized ability to synthesize and releas

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Novel Endothelin Receptors,. It was originally hypothesized that ET interacts with two subtypes of receptors to mediate its biological functions and subsequent cloning of two ET receptors confirmed the original hypothesis. As with any receptor, especially seven transmembrane G protein-coupled receptors, identification of rece

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