Titlebook: Endocrinology of Critical Disease; K. Patrick Ober Book 1997 Springer Science+Business Media New York 1997 Trauma.aldosterone.growth.growt

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Growth, Development, and Critical Disease,th the age, gender, and genetic background of the individual. Deviation from a previously defined pattern of growth or failure to undergo adolescent development at the appropriate time or tempo can often be the first clue to an underlying disease process.

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Pituitary Response to Stress,y via the hypothalamic—pituitary portal vessels (Fig. 1). There are PRL-releasing factors (PRF’s) as well. Disruption of the pituitary stalk leads to a moderate increase in PRL secretion as well as to decreased secretion of the other pituitary hormones.

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The Sympathoadrenomedullary Response to Critical Illness, ago, Claude Bernard theorized that the “milieu interne” must be maintained to preserve life. At about the same time, Walter Cannon introduced the term homeostasis to characterize “the coordinated physiological reactions that maintain the steady state of body” via the integrated cooperative activity

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The Adrenocortical Response to Critical Illness,hough the endocrine response to many forms of stress is relatively uniform, it is nevertheless complicated and comprehensive in scope. The adrenal cortex is a critical player in the endocrine response to major stress. Cortisol, the primary glucocorticoid secreted from the human adrenal gland, has lo

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Thyroid Response to Critical Illness,s in serum thyroid hormone indices, which, in certain instances, may minimize the catabolic impact of these events .. The initial step in this process is characterized by the development of the so-called low T. state where serum total and free T. values are reduced, but serum T. levels remain normal

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Pathophysiology of Water Metabolism During Critical Illness,ng periods of acute stress, such as critical illnesses. As might be expected of such important functions, many complex and overlapping physiological defense mechanisms have evolved to defend body fluid homeostasis during such periods. The efficacy of these mechanisms is attested to by the fact that

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Alterations in Fuel Metabolism in Critical Illness,His observation was subsequently confirmed by others who applied the terms “traumatic diabetes,” “diabetes of injury,” or “stress diabetes” to this state. It has been suggested that the evolutionary value of a hyperglycemic response to injury is to compensate for volume loss by promoting movement of