Titlebook: Endocrine Aspects of Successful Aging: Genes, Hormones and Lifestyles; Philippe Chanson,Jacques Epelbaum,Yves Christen Book 2004 Springer-

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Religion, Rebellion, Revolution the CREM (cAMP-responsive element modulator) gene, which encodes transcription factors responsive to activation of the cAMP signalling pathway. ICER (inducible cAMP early repressor) is a product of the CREM gene that is rhythmically expressed and participates in a transcriptional autoregulatory loo

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Defenders of the revolutionary traditionre not predictive of death. Further, the effect of growth hormone and testosterone replacement on quality of life has hardly been examined in the elderly population..Aging can be approached in two different ways: one can direct attention to the ensuing deficits or to the factors that play a protecti

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Polymorphisms and Male Ageing, longitudinal population studies demonstrate that the hormones with anabolic actions [e.g., testosterone (T), growth hormone, insulin-like growth factor (IGF)-1, dehydroepiandrosterone) fall progressively with ageing in healthy men, and various systemic diseases accelerate this process. Likewise, th

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Age-related Changes in the Regulation of the Hypothalamic-Pituitary Adrenal Axis: The Role of Perso effects of chronic activation of the HPA axis..Normal aging is accompanied by a number of changes in the regulation and activity of the HPA axis. Basal cortisol levels, especially at night around the time of the nadir, are increased, and feedback sensitivity to cortisol at different levels of the C

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Aging Myelin and Cognitive Decline: a Role for Steroids,that they are partly reversible. Abnormalities, breakdown and loss of myelin sheaths are reliable markers of the aging nervous system, which correlate with both chronological age and cognitive decline. Prolonged administration of progestins over one month to old male rats has been shown to reverse t

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Genetic Polymorphisms and Age-Related Diseases The Example of Osteoporosis,ns in early identification of at-risk subjects, prediction of response-to-treatment, and development of novel therapeutic options. We will discuss some developments in the field of the genetics of osteoporosis.