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Titlebook: Encephalopathy and Nitrogen Metabolism in Liver Failure; E. Anthony Jones,Alfred J. Meijer,Robert A. F. M. Book 2003 Kluwer Academic Publ

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发表于 2025-3-21 18:44:55 | 显示全部楼层 |阅读模式
书目名称Encephalopathy and Nitrogen Metabolism in Liver Failure
编辑E. Anthony Jones,Alfred J. Meijer,Robert A. F. M.
视频videohttp://file.papertrans.cn/310/309200/309200.mp4
图书封面Titlebook: Encephalopathy and Nitrogen Metabolism in Liver Failure;  E. Anthony Jones,Alfred J. Meijer,Robert A. F. M.  Book 2003 Kluwer Academic Publ
描述.Encephalopathy and Nitrogen Metabolism in Liver Failure. contains research reports and state-of-the-art reviews that were presented at the 11th International Symposium on Hepatic Encephalopathy and Nitrogen Metabolism in relation to liver disease, that was held in Amsterdam, The Netherlands from 30 May to 1 June, 2002. Themes covered include the metabolism of ammonia and amino acids in relation to liver disease, the assessment of hepatic encephalopathy (HE), minimal HE, animal models of HE, neuropsychiatric dysfunction in patients with chronic liver disease, the pathogenesis of HE, the astrocyte in liver disease, acute liver failure and cerebral edema, treatment of HE and artificial liver support systems. This volume provides an indispensable source of important new research findings and ideas for clinicians and biomedical scientists working in the field of HE and nitrogen metabolism in relation to liver disease.
出版日期Book 2003
关键词Amino acid; Glutamat; cortex; metabolism; physiology
版次1
doihttps://doi.org/10.1007/978-94-010-0159-5
isbn_softcover978-94-010-3967-3
isbn_ebook978-94-010-0159-5
copyrightKluwer Academic Publishers 2003
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Werkzeug- und Maschinen-Gerade,n of organs towards molecular and cell biology and, most recently, towards the integrated approach of systems biology. These developments have been associated with an enormous increase in knowledge, especially with regard to the role of nitrogenous compounds as signal molecules. Only a few aspects o
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Praxis der angiologischen Diagnostik. More specifically we describe new insights relating to the site of ammonia production after an UGI bleed, the catabolic consequences of an UGI bleed and a possible therapeutic intervention for preventing these consequences. The chapter is based upon a number of studies conducted by our group; some
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Praxis der technischen Direktwerbunggnitive impairment (mCI), and is usually used to diagnose mHE.. Event related evoked potentials (P300) have been proposed as a tool to improve the detection of mHE. in addition to psychometric tests. Another investigation that may have relevance to the diagnosis of mHE is spectral analysis of the EE
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,Infiltrationstherapie mit Lokalanästhesie,ts. It is not affected appreciably by training. CFFs change consistently in parallel with the evolution of HE after transjugular intrahepatic portal-systemic stent (TIPSS) placement. CFF determination is suitable for monitoring fluctuations in the severity of HE in response to precipitating factors
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