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Titlebook: Eicosanoids and Radiation; Peter Polgar Book 1988 Kluwer Academic Publishers, Boston 1988 cancer.cancer research.cell.cell culture.hormone

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Protection against Ionizing Radiation with Eicosanoids,apparent (1–3). The antagonistic properties of PGs have been implicated in a variety of symptoms resulting from exposure to ionizing radiation. Post irradiation increases in small bowel motility (4), diarrhea, flatulence, abdominal pain (5), mucositis (6), and esophagitis (7,8) have been attributed, in part, to excessive PG production.
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Finance and Sustainable Development,in production by a variety of tissues (3,4); however the precise mechanisms of such alterations and the role of prostaglandins in the patho-physiological effects of ionizing radiation have not been fully elucidated.
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Piyush Tiwari,Jyoti Rao,Jennifer Dayapparent (1–3). The antagonistic properties of PGs have been implicated in a variety of symptoms resulting from exposure to ionizing radiation. Post irradiation increases in small bowel motility (4), diarrhea, flatulence, abdominal pain (5), mucositis (6), and esophagitis (7,8) have been attributed, in part, to excessive PG production.
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The New International Economic Orderradiation are seen mainly in the tissues undergoing continual renewal such as mucosa, bone marrow, gonads etc., and the spectrum of damage ranging from progressive hypoplesia to total atrophy, depending on the dose. One of the late effects of radiation is the development of cancer.
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Radiation, Lipid Peroxidation and the Role of Oxygen Radicals in Eicosanoid Metabolism,ng, spleen, liver, skin and vasculature. While the precise role of PGs in the biological response to radiation is not fully understood, these alterations may be related to subsequent normal as well as pathophysiological responses.
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Nicholas Biekpe,Danny Cassimon,Karel Verbeke by radioimmunoassay for PGE. and PGE.. The goals of these studies have been to determine the role of eicosanoids 1) as markers of cytotoxicity, 2) in relationship to acute cell viability and long-term proliferative survival, 3) in relationship to receptors and cell/cell interactions, and 4) as radioprotective agents.
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