Titlebook: Effects of Mineral Dusts on Cells; Brooke T. Mossman,Raymond O. Bégin Conference proceedings 1989 Springer-Verlag Berlin Heidelberg 1989 A

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Communication and Synchronization, such as superoxide anion, neutrophil chemotactic factors, fibronectin (Wallaert, 1986). Here we have extended our study on macrophage secretory dysfunctions by the measurement of interleukin-1 and tumor necrosis factor alpha spontaneously released by alveolar macrophages.

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What is Data Quality and Why Should We Care?n appriopriate model has to be employed to provide a susceptible system for the effect of asbestos fibres and/or PAH on the immunity. F1(NZB*C57Bl/6) mouse hybrid was used. Due to the genetic trait inherited from NZB maternal strain, these mice show the immunological type of responsiveness (Theofilo

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1010-8793 studies. Mechanisms of cytotoxicity, mutagenicity and carcinogenicity. Oxidants, cytotoxicity and disease. Mechanisms of mineral-induced inflammation. Mechanis978-3-642-74205-7978-3-642-74203-3Series ISSN 1010-8793

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Injurious Effects of Mineral Dust-elicited Bronchoalveolar Leukocytes on Epithelial Cells ,: The Rold by the more appropriate inhalation route to coalmine dust, with the regard to causing epithelial injury and proteolytic degradation of fibronectin. In order to study the role of the extracellular matrix in epithelial injury ., we have examined the ability of exogenous protease and mineral dust-eli

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A Comparison of the , and , Toxicity of Fibrous and Non-Fibrous Silicon Nitride the . evaluation that the non-fibrous Si.N. was relatively inert. Alpha-quartz induced significant pulmonary toxicity including fibrosis, edema, and coalescing granulomas. The fibrous Si.N. produced minimal pulmonary changes because physical limitations associated with instilling the fibrous Si.N.

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