Titlebook: Early Osteoarthritis; State-of-the-Art App Christian Lattermann,Henning Madry,Elizaveta Kon Book 2022 ISAKOS 2022 Articular cartilage.chron

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Early OA Following Synovial Joint Fractureit possible to assess the efficacy of treatments intended to reduce PTOA risk. In vitro and in vivo investigations of joint biologic responses to excessive loads show that reactive oxygen species (ROS) produced by chondrocytes cause mitochondrial damage and dysfunction, chondrocyte death, matrix deg

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Inflammation After Anterior Cruciate Ligament Injuryences in individualized inflammatory responses are important to consider in treatment. Moreover, ACL reconstruction surgery presents a second and potentially larger insult to the joint resulting in the re-initiation of inflammation and an increase in cartilage breakdown. This “second hit” triggers a

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Fat-Derived Stem Cellssubcutaneous adipose tissue that represents therefore a plentiful source of the MSC/pericytes. Within fat, adipose stem cells (ASCs) together with other cell types are embedded in a rich vascular network and constitute the “stroma” or “stromal vascular fraction” (SVF). ASCs possess similar features

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