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Titlebook: ESKAPE Pathogens; Detection, Mechanism Siddhardha Busi,Ram Prasad Book 2024 The Editor(s) (if applicable) and The Author(s), under exclusiv

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Antibiotic Resistance Profile and Detection in ESKAPE Pathogens,ory antibiotic concentration. Increased antibiotic resistance of bacterial pathogens and their spread in the clinical sector, industry and environment is a grave threat to therapeutic applications and healthcare globally. Owing to uncontrolled spread, antibiotic resistance loomed as a global “immine
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Mechanistic Understanding of Antibiotic Resistance in ESKAPE Pathogens,these pathogens that have acquired resistance to commonly accessible drugs. Antibiotic resistance is a complex process involving genetic and epigenetic factors, and horizontal gene transfer is the most prevalent resistance mechanism in these pathogens. The propagation of multiple antibiotic-resistan
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Nucleic Acid Amplification and Molecular Diagnostic Techniques in the Detection of ESKAPE Bacterialocomial infections and also have the potential to develop drug resistance owing to their ability to “escape” the outcome action of antibiotics. The current chapter gives an overview of the ESKAPE pathogens, diseases associated with these pathogens, available methods to diagnose these pathogens with
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Biochemical, Molecular, and Computational Techniques for the Determination of Virulence Factors of ics raised the emergence of multidrug-resistant (MDR) and extensively drug-resistant bacteria (XDR) that ultimately cause treatment failures. Among all the clinical pathogens the World Health Organization (WHO) has reported the most common critical pathogen in hospital settings. The most listed six
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Virulence Factors and Biofilm Components: Synthesis, Structure, Function and Inhibitors,ounds and also immunocompromised individuals. Diverse resistance mechanisms possessed by the bacterium make them to overcome the effect of various antimicrobial agents. . possess a plenty of virulence determinants, specifically biofilm, and triggers resistance, which is controlled by cellular commun
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