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Titlebook: Dopamine in the Gut; Jin-Xia Zhu Book 2021 Springer Nature Singapore Pte Ltd. 2021 Dopamine.dopamine receptor.brain-gut axis.gastrointesti

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Dopamine Receptors in the Gastrointestinal Tract,ange of physiological roles in the GI tract after binding to the DARs and/or the adrenergic receptors, which includes protection of GI mucosa, regulation of GI motility, secretion and absorption, etc. Here we predominately review the distribution of DARs on the GI mucosa, muscularis externa, and enteric nervous system.
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Ungleichheitskonflikte in Europaer is densely populated by a myriad of distinct nerve fibers, endocrine cells, and immune cells that sense, integrate, and respond to multiple environmental cues. In this chapter, we focus on the effects of DA on the GI secretion and absorption, ion transport, mucosal barrier, blood flow, and mucosal regulation.
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https://doi.org/10.1007/978-3-658-22245-1cular monoamine transporter 2 (VMAT2) and D.R have become the research hot spots to look for the therapeutic targets and develop the pancreatic islets imaging probes used for monitoring β-cell mass morphology and physiology.
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Book 2021s a dopaminergic neurodegenerative disorder, and affected patients often show early signs of gastrointestinal disorders at initial stages of the disease. Apart from the changes that occur in emotion, movement behaviors, learning and memory, metabolism etc., dopamine has also been demonstrated to pla
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Eurokrise und soziale Ungleichheitmode of DA in GI motility regulation, via acting on smooth muscle cells, intrinsic neurons, and extrinsic neurons. In pathological states, such as Parkinson’s disease (PD), we summarize the symptoms and underlying mechanism of abnormal GI motility in PD patients. Furthermore, we review GI motility function in several PD animal models.
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Dopamine and Gastrointestinal Motility,mode of DA in GI motility regulation, via acting on smooth muscle cells, intrinsic neurons, and extrinsic neurons. In pathological states, such as Parkinson’s disease (PD), we summarize the symptoms and underlying mechanism of abnormal GI motility in PD patients. Furthermore, we review GI motility function in several PD animal models.
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