Titlebook: Dietary Fats, Prostanoids and Arterial Thrombosis; Gerard Hornstra Book 1982 Martinus Nijhoff Publishers, The Hague 1982 blood.metabolism.

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Developments in Hematology and Immunologyhttp://image.papertrans.cn/d/image/278577.jpg

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Book 1982oronary arteries in up to 90% of subjects with acute myocardial infaction. In this volume Dr. Hornstra, who has occupied himself with thrombosis research with never-failing enthusiasm, great skill and critical approach for the last fifteen years, tries to establish possible links between lipid metab

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d in the coronary arteries in up to 90% of subjects with acute myocardial infaction. In this volume Dr. Hornstra, who has occupied himself with thrombosis research with never-failing enthusiasm, great skill and critical approach for the last fifteen years, tries to establish possible links between lipid metab978-94-009-7578-1978-94-009-7576-7

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Johannes Köper,Hans Jürgen Zarembased after vessel-trauma, rupture of an atherosclerotic plaque, etc. Adhered platelets release some of their constituents, such as adenine nucleotides, serotonin, Ca. and adrenaline. Adenosine diphosphate (ADP), made available by this release reaction, causes blood platelets, to aggregate: passing pl

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Qualitätssicherung durch Prozeßorientierungcal studies is rather limited, however, because they only detect associations and do not prove any cause-effect relationships. For this purpose, prospective clinical trials have been performed, which all indicate a favourable effect on atherosclerotic complications of a diet low in saturated fats an

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interpose

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Relationship between Dietary Fat Type, Platelet Fatty Acid Composition and Eicosanoid Formation by ., PGI.) production in a strikingly similar way: although the . of the different compounds produced were greatly affected by the type of dietary fat, the . between both products remained the same. This suggests that the type of dietary fat affects a common factor, which is as important for TxA.- as it is for PGI.-formation.

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https://doi.org/10.1007/978-3-642-57974-5rterial thrombogenesis might be mediated by prostaglandins and/or related compounds. In order to investigate the possible role of these compounds in arterial thrombus formation, we decided to investigate the effect of essential fatty acid deficiency [391] which is associated with a low PG production in tissues [197, 800].