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Titlebook: Diet and Cancer; Molecular Mechanisms American Institute for Cancer Research Book 1995 The Editor(s) (if applicable) and The Author(s), und

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楼主: HEM
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Vitamin A Chemoprevention of Lung Cancer,rocesses of inflammation and carcinogenesis are linked. We have hypothesized that 1) the vitamin A intervention may reduce both bronchial metaplasia and lung inflammation, 2) the mechanism of this effect may be through modulation of relevant pulmonary cytokines, growth factors, and/or oncogenes, and
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Vitamin D and Prostate Cancer,ed death in U.S. men after lung cancer. Mortality rates from prostate cancer appear to be on the rise. From 1970 to 1990 the age-adjusted mortality rate from prostate cancer increased approximately 7 % among U.S. caucasians. Since prostate cancer rates increase with age, as the longevity of the popu
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Dietary Effects on Gene Expression in Mammary Tumorigenesis,idemiological data in humans have been for the most part based on comparison of breast cancer incidence and fat consumption in different countries worldwide.. Recently, however, two prospective epidemiological studies. involving a large cohort of women have suggested a lack of evidence for a relatio
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Progressive Loss of Sensitivity to Growth Control by Retinoic Acid and Transforming Growth Factor-Bthat no longer require EGF and BPE for proliferation, are selected from HKc/HPV16.. Next, sublines that no longer respond to calcium and serum-induced differentiation, HKc/DR, are selected from HKc/GFI.. In the final step, malignant conversion of HKc/DR is achieved by transfection with viral Harvey
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Fish Oil and Cell Proliferation Kinetics in a Mammary Carcinoma Tumor Model, notion that, within a given cell type, DNA replication time (S phase duration) is constant (4–6). Extensive evidence also supports an on/off mechanism of cell cycle regulation at the level of entry into the S phase (3)..In this report, we present evidence showing, for the first time, that the S pha
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